Correction: GPR119 agonist enhances gefitinib responsiveness through lactate-mediatedinhibition of autophagy
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1756-9966
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Correction: GPR119 agonist enhances gefitinib responsiveness through lactate-mediatedinhibition of autophagy ; volume:41 ; number:1 ; day:17 ; month:5 ; year:2022 ; pages:1 ; date:12.2022
Journal of experimental & clinical cancer research ; 41, Heft 1 (17.5.2022), 1, 12.2022
- Urheber
-
Im, Ji Hye
Kang, Keon Wook
Kim, Sun Young
Kim, Yoon Gyoon
An, Yong Jin
Park, Sunghyouk
Jung, Byung Hwa
Choi, Song-Yi
Lee, Jin-Sun
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s13046-022-02388-0
- URN
-
urn:nbn:de:101:1-2022072306281210694038
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:25 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Im, Ji Hye
- Kang, Keon Wook
- Kim, Sun Young
- Kim, Yoon Gyoon
- An, Yong Jin
- Park, Sunghyouk
- Jung, Byung Hwa
- Choi, Song-Yi
- Lee, Jin-Sun
- SpringerLink (Online service)
Ähnliche Objekte (12)
GPR119 agonist enhances gefitinib responsiveness through lactate-mediated inhibition of autophagy
Gefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coatingGefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coating
A New Synthesis of Gefitinib
Curcumin overcome primary gefitinib resistance in non-small-cell lung cancer cells through inducing autophagy-related cell death
The mechanism of m6A methyltransferase METTL3-mediated autophagy in reversing gefitinib resistance in NSCLC cells by β-elemene
In vitro-Untersuchungen zur Wirksamkeit von Erlotinib, Gefitinib und Cetuximab bei der Behandlung des Kopf-Hals-Karzinoms : = In-vitro study on erlotinib, gefitinib and cetuximab responsiveness in head and neck cancer cell lines
Exploring the relationship between autophagy and Gefitinib resistance in NSCLC by silencing PDLIM5 using ultrasound-targeted microbubble destruction technology
Clinical benefit of readministration of gefitinib for initial gefitinib-responders with non-small cell lung cancer
Drittlinientherapie mit Gefitinib beim nicht-kleinzelligen Bronchialkarzinom
SFRP4 promotes autophagy and blunts FSH responsiveness through inhibition of AKT signaling in ovarian granulosa cells
Dosis-Wirkungsbeziehungen von Gefitinib in einem humanen Lungentumormodell
Radiation myelitis after hypofractionated radiotherapy with concomitant gefitinib
GPR119 agonist enhances gefitinib responsiveness through lactate-mediated inhibition of autophagy
Gefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coatingGefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coating
A New Synthesis of Gefitinib
Curcumin overcome primary gefitinib resistance in non-small-cell lung cancer cells through inducing autophagy-related cell death
The mechanism of m6A methyltransferase METTL3-mediated autophagy in reversing gefitinib resistance in NSCLC cells by β-elemene
In vitro-Untersuchungen zur Wirksamkeit von Erlotinib, Gefitinib und Cetuximab bei der Behandlung des Kopf-Hals-Karzinoms : = In-vitro study on erlotinib, gefitinib and cetuximab responsiveness in head and neck cancer cell lines
Exploring the relationship between autophagy and Gefitinib resistance in NSCLC by silencing PDLIM5 using ultrasound-targeted microbubble destruction technology
Clinical benefit of readministration of gefitinib for initial gefitinib-responders with non-small cell lung cancer
Drittlinientherapie mit Gefitinib beim nicht-kleinzelligen Bronchialkarzinom
SFRP4 promotes autophagy and blunts FSH responsiveness through inhibition of AKT signaling in ovarian granulosa cells
Dosis-Wirkungsbeziehungen von Gefitinib in einem humanen Lungentumormodell
Radiation myelitis after hypofractionated radiotherapy with concomitant gefitinib
GPR119 agonist enhances gefitinib responsiveness through lactate-mediated inhibition of autophagy
Gefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coatingGefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coating
A New Synthesis of Gefitinib
Curcumin overcome primary gefitinib resistance in non-small-cell lung cancer cells through inducing autophagy-related cell death
The mechanism of m6A methyltransferase METTL3-mediated autophagy in reversing gefitinib resistance in NSCLC cells by β-elemene
In vitro-Untersuchungen zur Wirksamkeit von Erlotinib, Gefitinib und Cetuximab bei der Behandlung des Kopf-Hals-Karzinoms : = In-vitro study on erlotinib, gefitinib and cetuximab responsiveness in head and neck cancer cell lines
Exploring the relationship between autophagy and Gefitinib resistance in NSCLC by silencing PDLIM5 using ultrasound-targeted microbubble destruction technology
Clinical benefit of readministration of gefitinib for initial gefitinib-responders with non-small cell lung cancer
Drittlinientherapie mit Gefitinib beim nicht-kleinzelligen Bronchialkarzinom
SFRP4 promotes autophagy and blunts FSH responsiveness through inhibition of AKT signaling in ovarian granulosa cells
Dosis-Wirkungsbeziehungen von Gefitinib in einem humanen Lungentumormodell