GPR119 agonist enhances gefitinib responsiveness through lactate-mediated inhibition of autophagy
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1756-9966
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
GPR119 agonist enhances gefitinib responsiveness through lactate-mediated inhibition of autophagy ; volume:37 ; number:1 ; day:29 ; month:11 ; year:2018 ; pages:1-15 ; date:12.2018
Journal of experimental & clinical cancer research ; 37, Heft 1 (29.11.2018), 1-15, 12.2018
- Classification
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Medizin, Gesundheit
- Creator
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Im, Ji Hye
Kang, Keon Wook
Kim, Sun Young
Kim, Yoon Gyoon
An, Yong Jin
Park, Sunghyouk
Jeong, Byung Hwa
Choi, Song-Yi
Lee, Jin-Sun
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1186/s13046-018-0949-2
- URN
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urn:nbn:de:101:1-2019011622065148186328
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:53 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Im, Ji Hye
- Kang, Keon Wook
- Kim, Sun Young
- Kim, Yoon Gyoon
- An, Yong Jin
- Park, Sunghyouk
- Jeong, Byung Hwa
- Choi, Song-Yi
- Lee, Jin-Sun
- SpringerLink (Online service)
Other Objects (12)
Correction: GPR119 agonist enhances gefitinib responsiveness through lactate-mediatedinhibition of autophagy
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Clinical benefit of readministration of gefitinib for initial gefitinib-responders with non-small cell lung cancer
Drittlinientherapie mit Gefitinib beim nicht-kleinzelligen Bronchialkarzinom
SFRP4 promotes autophagy and blunts FSH responsiveness through inhibition of AKT signaling in ovarian granulosa cells
Dosis-Wirkungsbeziehungen von Gefitinib in einem humanen Lungentumormodell
Radiation myelitis after hypofractionated radiotherapy with concomitant gefitinib
Correction: GPR119 agonist enhances gefitinib responsiveness through lactate-mediatedinhibition of autophagy
Gefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coatingGefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coating
A New Synthesis of Gefitinib
Curcumin overcome primary gefitinib resistance in non-small-cell lung cancer cells through inducing autophagy-related cell death
The mechanism of m6A methyltransferase METTL3-mediated autophagy in reversing gefitinib resistance in NSCLC cells by β-elemene
In vitro-Untersuchungen zur Wirksamkeit von Erlotinib, Gefitinib und Cetuximab bei der Behandlung des Kopf-Hals-Karzinoms : = In-vitro study on erlotinib, gefitinib and cetuximab responsiveness in head and neck cancer cell lines
Exploring the relationship between autophagy and Gefitinib resistance in NSCLC by silencing PDLIM5 using ultrasound-targeted microbubble destruction technology
Clinical benefit of readministration of gefitinib for initial gefitinib-responders with non-small cell lung cancer
Drittlinientherapie mit Gefitinib beim nicht-kleinzelligen Bronchialkarzinom
SFRP4 promotes autophagy and blunts FSH responsiveness through inhibition of AKT signaling in ovarian granulosa cells
Dosis-Wirkungsbeziehungen von Gefitinib in einem humanen Lungentumormodell
Radiation myelitis after hypofractionated radiotherapy with concomitant gefitinib
Correction: GPR119 agonist enhances gefitinib responsiveness through lactate-mediatedinhibition of autophagy
Gefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coatingGefitinib/gefitinib microspheres loaded polyurethane constructs as drug-eluting stent coating
A New Synthesis of Gefitinib
Curcumin overcome primary gefitinib resistance in non-small-cell lung cancer cells through inducing autophagy-related cell death
The mechanism of m6A methyltransferase METTL3-mediated autophagy in reversing gefitinib resistance in NSCLC cells by β-elemene
In vitro-Untersuchungen zur Wirksamkeit von Erlotinib, Gefitinib und Cetuximab bei der Behandlung des Kopf-Hals-Karzinoms : = In-vitro study on erlotinib, gefitinib and cetuximab responsiveness in head and neck cancer cell lines
Exploring the relationship between autophagy and Gefitinib resistance in NSCLC by silencing PDLIM5 using ultrasound-targeted microbubble destruction technology
Clinical benefit of readministration of gefitinib for initial gefitinib-responders with non-small cell lung cancer
Drittlinientherapie mit Gefitinib beim nicht-kleinzelligen Bronchialkarzinom
SFRP4 promotes autophagy and blunts FSH responsiveness through inhibition of AKT signaling in ovarian granulosa cells
Dosis-Wirkungsbeziehungen von Gefitinib in einem humanen Lungentumormodell