Early life sleep disruption potentiates lasting sex-specific changes in behavior in genetically vulnerable Shank3 heterozygous autism model mice
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2040-2392
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
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Early life sleep disruption potentiates lasting sex-specific changes in behavior in genetically vulnerable Shank3 heterozygous autism model mice ; volume:13 ; number:1 ; day:29 ; month:8 ; year:2022 ; pages:1-19 ; date:12.2022
Molecular autism ; 13, Heft 1 (29.8.2022), 1-19, 12.2022
- Urheber
-
Lord, Julia S.
Gay, Sean M.
Harper, Kathryn M.
Nikolova, Viktoriya D.
Smith, Kirsten M.
Moy, Sheryl S.
Diering, Graham H.
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
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10.1186/s13229-022-00514-5
- URN
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urn:nbn:de:101:1-2022111621062505074139
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:31 MESZ
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Beteiligte
- Lord, Julia S.
- Gay, Sean M.
- Harper, Kathryn M.
- Nikolova, Viktoriya D.
- Smith, Kirsten M.
- Moy, Sheryl S.
- Diering, Graham H.
- SpringerLink (Online service)
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Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Pre-implantation alcohol exposure induces lasting sex-specific DNA methylation programming errors in the developing forebrain
SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids
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Wittgenstein's lasting significance
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Expanding the Nude SCID/CID Phenotype Associated with FOXN1 Homozygous, Compound Heterozygous, or Heterozygous Mutations
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
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SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids
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New insights into SHANK3-associated diseases and treatment options
A lasting peace
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Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
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