Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2040-2392
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice ; volume:5 ; number:1 ; day:25 ; month:4 ; year:2014 ; pages:1-14 ; date:12.2014
Molecular autism ; 5, Heft 1 (25.4.2014), 1-14, 12.2014
- Creator
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Wang, Xiaoming
Xu, Qiong
Bey, Alexandra L.
Lee, Yoonji
Jiang, Yong-hui
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/2040-2392-5-30
- URN
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urn:nbn:de:101:1-2021081620331448842508
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:48 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wang, Xiaoming
- Xu, Qiong
- Bey, Alexandra L.
- Lee, Yoonji
- Jiang, Yong-hui
- SpringerLink (Online service)
Other Objects (12)
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Microglia unravel immune-associated alterations in Shank3 deficiency
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New insights into SHANK3-associated diseases and treatment options
Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
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ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma
Haploinsufficiency of Shank3 increases the orientation selectivity of V1 neurons
Brain region-specific disruption of Shank3 in mice reveals a dissociation for cortical and striatal circuits in autism-related behaviors
Ultrastructural analyses in the hippocampus CA1 field in Shank3-deficient mice
SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids
Microglia unravel immune-associated alterations in Shank3 deficiency
Funktionelle Analyse von SHANK3-Mutationen assoziiert mit Synaptopathien
New insights into SHANK3-associated diseases and treatment options
Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
Zweistufige Verflüssigung einer chinesischen Steinkohle
Normalized multi-bump solutions for saturable Schrödinger equations
Aubry-Mather sets in semilinear asymmetric Duffing equations
ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma
Haploinsufficiency of Shank3 increases the orientation selectivity of V1 neurons
Brain region-specific disruption of Shank3 in mice reveals a dissociation for cortical and striatal circuits in autism-related behaviors
Ultrastructural analyses in the hippocampus CA1 field in Shank3-deficient mice
SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids
Microglia unravel immune-associated alterations in Shank3 deficiency
Funktionelle Analyse von SHANK3-Mutationen assoziiert mit Synaptopathien
New insights into SHANK3-associated diseases and treatment options
Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
Zweistufige Verflüssigung einer chinesischen Steinkohle
Normalized multi-bump solutions for saturable Schrödinger equations
Aubry-Mather sets in semilinear asymmetric Duffing equations
ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma
Haploinsufficiency of Shank3 increases the orientation selectivity of V1 neurons
Brain region-specific disruption of Shank3 in mice reveals a dissociation for cortical and striatal circuits in autism-related behaviors