Inhibition of eIF2α dephosphorylation inhibits ErbB2-induced deregulation of mammary acinar morphogenesis
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2121
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Inhibition of eIF2α dephosphorylation inhibits ErbB2-induced deregulation of mammary acinar morphogenesis ; volume:10 ; number:1 ; day:15 ; month:9 ; year:2009 ; pages:1-14 ; date:12.2009
BMC cell biology ; 10, Heft 1 (15.9.2009), 1-14, 12.2009
- Creator
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Sequeira, Sharon J.
Wen, Huei Chi
Avivar-Valderas, Alvaro
Farias, Eduardo F.
Aguirre-Ghiso, Julio A.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/1471-2121-10-64
- URN
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urn:nbn:de:101:1-2021080921174660113784
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:57 AM CEST
Data provider
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Associated
- Sequeira, Sharon J.
- Wen, Huei Chi
- Avivar-Valderas, Alvaro
- Farias, Eduardo F.
- Aguirre-Ghiso, Julio A.
- SpringerLink (Online service)
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Pỉexin-B2 controls mitotic spindle orientation during epithelial morphogenesis and repair
C/EBPbeta-2 confers EGF-independent growth and disrupts the normal acinar architecture of human mammary epithelial cells
Focal adhesion kinase contributes to proliferative potential of ErbB2 mammary tumour cells but is dispensable for ErbB2 mammary tumour induction in vivo
LOXL2 induces aberrant acinar morphogenesis via ErbB2 signaling
Mammary epithelial morphogenesis in 3D combinatorial microenvironments
The Hippo Signaling Transducer TAZ Regulates Mammary Gland Morphogenesis and Carcinogen-induced Mammary Tumorigenesis
RARα1 control of mammary gland ductal morphogenesis and wnt1-tumorigenesis
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ErbB3 is required for ductal morphogenesis in the mouse mammary gland
Interplay of the transcription factor MRTF-A and matrix stiffness controls mammary acinar structure and protrusion formation
Pỉexin-B2 controls mitotic spindle orientation during epithelial morphogenesis and repair
C/EBPbeta-2 confers EGF-independent growth and disrupts the normal acinar architecture of human mammary epithelial cells