PLA2G7/PAF-AH as a protective factor of BRCA1 mutant breast cancer and ovarian cancer and negatively regulate the Wnt signaling pathway
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
München, Ludwig-Maximilians-Universität, Dissertation, 2022
- Keyword
-
Cancer
Breast--Cancer
- Event
-
Veröffentlichung
- (where)
-
München
- (who)
-
Universitätsbibliothek der Ludwig-Maximilians-Universität
- (when)
-
2022
- Creator
- Contributor
-
Jeschke, Udo
- DOI
-
10.5282/edoc.29605
- URN
-
urn:nbn:de:bvb:19-296055
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:43 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Liao, Yue
- Jeschke, Udo
- Universitätsbibliothek der Ludwig-Maximilians-Universität
Time of origin
- 2022
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Ovarian cancer susceptibility alleles and risk of ovarian cancer in BRCA1 and BRCA2 mutation carriers
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Common variants of the BRCA1 wild-type allele modify the risk of breast cancer in BRCA1 mutation carriers
Associations of common breast cancer susceptibility alleles with risk of breast cancer subtypes in BRCA1 and BRCA2 mutation carriers
Constitutive promoter methylation of BRCA1 and RAD51C in patients with familial ovarian cancer and early-onset sporadic breast cancer
Genetic versus epigenetic BRCA1 silencing pathways: clinical effects in primary ovarian cancer patients
Thyroid hormone receptors predict prognosis in BRCA1 associated breast cancer in opposing ways
Ovarian cancer-cell pericellular hyaluronan deposition negatively impacts prognosis of ovarian cancer patients
Monozygotic twins discordant for constitutive BRCA1 promoter methylation, childhood cancer and secondary cancer
PLA2G7/PAF-AH as potential negative regulator of the Wnt signaling pathway mediates protective effects in BRCA1 mutant breast cancer
Ovarian cancer susceptibility alleles and risk of ovarian cancer in BRCA1 and BRCA2 mutation carriers
Negatively-charged supported lipid bilayers regulate neuronal adhesion and outgrowth
Loss of BRCA1 promoter hypermethylation in recurrent high-grade ovarian cancer
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Common variants of the BRCA1 wild-type allele modify the risk of breast cancer in BRCA1 mutation carriers
Associations of common breast cancer susceptibility alleles with risk of breast cancer subtypes in BRCA1 and BRCA2 mutation carriers
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