Mucopolysaccharidosis IIIB, a lysosomal storage disease, triggers a pathogenic CNS autoimmune response

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1742-2094

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Mucopolysaccharidosis IIIB, a lysosomal storage disease, triggers a pathogenic CNS autoimmune response ; volume:7 ; number:1 ; day:16 ; month:7 ; year:2010 ; pages:1-8 ; date:12.2010
Journal of neuroinflammation ; 7, Heft 1 (16.7.2010), 1-8, 12.2010

Creator: Killedar, Smruti

Contributor: DiRosario, Julianne
Divers, Erin
Popovich, Phillip G.
McCarty, Douglas M.
Fu, Haiyan
SpringerLink (Online service)

DOI: 10.1186/1742-2094-7-39

URN: urn:nbn:de:1111-2016032327951

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:45 AM CEST

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Killedar, Smruti
DiRosario, Julianne
Divers, Erin
Popovich, Phillip G.
McCarty, Douglas M.
Fu, Haiyan
SpringerLink (Online service)

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Mucopolysaccharidosis IIIB, a lysosomal storage disease, triggers a pathogenic CNS autoimmune response

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Other Objects (12)

Methylmalonic acidemia triggers lysosomal-autophagy dysfunctions

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Early lysosomal maturation deficits in microglia triggers enhanced lysosomal activity in other brain cells of progranulin knockout mice

Adaptations of autophagy and lysosomal systems upon membrane damage and pathogenic invasion

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IIIb.

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EGFR activation triggers cellular hypertrophy and lysosomal disease in NAGLU-depleted cardiomyoblasts, mimicking the hallmarks of mucopolysaccharidosis IIIB

Inducin Triggers LC3‐Lipidation and ESCRT‐Mediated Lysosomal Membrane Repair

Early lysosomal maturation deficits in microglia triggers enhanced lysosomal activity in other brain cells of progranulin knockout mice

Adaptations of autophagy and lysosomal systems upon membrane damage and pathogenic invasion

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IIIb.

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EGFR activation triggers cellular hypertrophy and lysosomal disease in NAGLU-depleted cardiomyoblasts, mimicking the hallmarks of mucopolysaccharidosis IIIB

Inducin Triggers LC3‐Lipidation and ESCRT‐Mediated Lysosomal Membrane Repair

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