- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2058-7716
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Hyperglycemia potentiates a shift from apoptosis to RIP1-dependent necroptosis ; volume:4 ; number:1 ; day:10 ; month:5 ; year:2018 ; pages:1-14 ; date:12.2018
Cell death discovery ; 4, Heft 1 (10.5.2018), 1-14, 12.2018
- Creator
-
McCaig, William D.
- Contributor
-
Patel, Payal S.
Sosunov, Sergey A.
Shakerley, Nicole L.
Smiraglia, Tori A.
Craft, Miranda M.
Walker, Katharine M.
Deragon, Matthew A.
Ten, Vadim S.
LaRocca, Timothy J.
SpringerLink (Online service)
- DOI
-
10.1038/s41420-018-0058-1
- URN
-
urn:nbn:de:101:1-2018071514155133443446
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:27 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- McCaig, William D.
- Patel, Payal S.
- Sosunov, Sergey A.
- Shakerley, Nicole L.
- Smiraglia, Tori A.
- Craft, Miranda M.
- Walker, Katharine M.
- Deragon, Matthew A.
- Ten, Vadim S.
- LaRocca, Timothy J.
- SpringerLink (Online service)
Other Objects (12)
Mitochondrial ROS prime the hyperglycemic shift from apoptosis to necroptosis
Apoptosis, autophagy, necroptosis, and cancer metastasis
Necroptosis and apoptosis in the primate ovary
Interferon‐mediated repression of miR ‐324‐5p potentiates necroptosis to facilitate antiviral defense
TRAF2 inhibits TRAIL- and CD95L-induced apoptosis and necroptosis
Myeloid-derived miR-6236 potentiates adipocyte insulin signaling and prevents hyperglycemia during obesity
Sheng Mai San protects H9C2 cells against hyperglycemia-induced apoptosis
Smac mimetic-induced necroptosis bypasses apoptosis resistance in acute myeloid leukemia
Apoptosis, necrosis and necroptosis: cell death regulation in the intestinal epithelium
PANoptosis: bridging apoptosis, pyroptosis, and necroptosis in cancer progression and treatment
RIPK1 can mediate apoptosis in addition to necroptosis during embryonic development
Caspase-8 is the molecular switch for apoptosis, necroptosis and pyroptosis
Mitochondrial ROS prime the hyperglycemic shift from apoptosis to necroptosis
Apoptosis, autophagy, necroptosis, and cancer metastasis
Necroptosis and apoptosis in the primate ovary
Interferon‐mediated repression of miR ‐324‐5p potentiates necroptosis to facilitate antiviral defense
TRAF2 inhibits TRAIL- and CD95L-induced apoptosis and necroptosis
Myeloid-derived miR-6236 potentiates adipocyte insulin signaling and prevents hyperglycemia during obesity
Sheng Mai San protects H9C2 cells against hyperglycemia-induced apoptosis
Smac mimetic-induced necroptosis bypasses apoptosis resistance in acute myeloid leukemia
Apoptosis, necrosis and necroptosis: cell death regulation in the intestinal epithelium
PANoptosis: bridging apoptosis, pyroptosis, and necroptosis in cancer progression and treatment
RIPK1 can mediate apoptosis in addition to necroptosis during embryonic development
Caspase-8 is the molecular switch for apoptosis, necroptosis and pyroptosis
Mitochondrial ROS prime the hyperglycemic shift from apoptosis to necroptosis
Apoptosis, autophagy, necroptosis, and cancer metastasis
Necroptosis and apoptosis in the primate ovary
Interferon‐mediated repression of miR ‐324‐5p potentiates necroptosis to facilitate antiviral defense
TRAF2 inhibits TRAIL- and CD95L-induced apoptosis and necroptosis
Myeloid-derived miR-6236 potentiates adipocyte insulin signaling and prevents hyperglycemia during obesity
Sheng Mai San protects H9C2 cells against hyperglycemia-induced apoptosis
Smac mimetic-induced necroptosis bypasses apoptosis resistance in acute myeloid leukemia
Apoptosis, necrosis and necroptosis: cell death regulation in the intestinal epithelium
PANoptosis: bridging apoptosis, pyroptosis, and necroptosis in cancer progression and treatment
RIPK1 can mediate apoptosis in addition to necroptosis during embryonic development