Protective effect of PDE4B subtype-specific inhibition in an App knock-in mouse model for Alzheimer’s disease

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Protective effect of PDE4B subtype-specific inhibition in an App knock-in mouse model for Alzheimer’s disease ; day:23 ; month:3 ; year:2024 ; pages:1-10
Neuropsychopharmacology ; (23.3.2024), 1-10

Creator: Armstrong, Paul
Güngör, Hüseyin
Anongjanya, Pariya
Tweedy, Clare
Parkin, Edward
Johnston, Jamie
Carr, Ian M.
Dawson, Neil
Clapcote, Steven J.

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41386-024-01852-z

URN: urn:nbn:de:101:1-2405270913562.283736081012

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:53 AM CEST

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Armstrong, Paul
Güngör, Hüseyin
Anongjanya, Pariya
Tweedy, Clare
Parkin, Edward
Johnston, Jamie
Carr, Ian M.
Dawson, Neil
Clapcote, Steven J.
SpringerLink (Online service)

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Protective effect of PDE4B subtype-specific inhibition in an App knock-in mouse model for Alzheimer’s disease

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Other Objects (12)

Generation of App knock-in mice reveals deletion mutations protective against Alzheimer’s disease-like pathology

Neuronal transcriptome, tau and synapse loss in Alzheimer’s knock-in mice require prion protein

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Brain region–specific lipid alterations in the PLB4 hBACE1 knock-in mouse model of Alzheimer’s disease

Knock, Knock, Knockin’ on Tranformativeness’ Doors

Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models

Age-related changes in brain phospholipids and bioactive lipids in the APP knock-in mouse model of Alzheimer’s disease

Microglial-associated responses to comorbid amyloid pathology and hyperhomocysteinemia in an aged knock-in mouse model of Alzheimer’s disease

Impact of hyperhomocysteinemia and different dietary interventions on cognitive performance in a knock-in mouse model for Alzheimer’s disease

Comprehensive behavioral characterization of an APP/PS-1 double knock-in mouse model of Alzheimer's disease

Impairment in novelty-promoted memory via behavioral tagging and capture before apparent memory loss in a knock-in model of Alzheimer’s disease

Vascular and parenchymal amyloid pathology in an Alzheimer disease knock-in mouse model: interplay with cerebral blood flow

Generation of App knock-in mice reveals deletion mutations protective against Alzheimer’s disease-like pathology

Neuronal transcriptome, tau and synapse loss in Alzheimer’s knock-in mice require prion protein

Knock-in models related to Alzheimer’s disease: synaptic transmission, plaques and the role of microglia

Brain region–specific lipid alterations in the PLB4 hBACE1 knock-in mouse model of Alzheimer’s disease

Knock, Knock, Knockin’ on Tranformativeness’ Doors

Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models

Age-related changes in brain phospholipids and bioactive lipids in the APP knock-in mouse model of Alzheimer’s disease

Microglial-associated responses to comorbid amyloid pathology and hyperhomocysteinemia in an aged knock-in mouse model of Alzheimer’s disease

Impact of hyperhomocysteinemia and different dietary interventions on cognitive performance in a knock-in mouse model for Alzheimer’s disease

Comprehensive behavioral characterization of an APP/PS-1 double knock-in mouse model of Alzheimer's disease

Impairment in novelty-promoted memory via behavioral tagging and capture before apparent memory loss in a knock-in model of Alzheimer’s disease

Vascular and parenchymal amyloid pathology in an Alzheimer disease knock-in mouse model: interplay with cerebral blood flow

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Neuronal transcriptome, tau and synapse loss in Alzheimer’s knock-in mice require prion protein

Knock-in models related to Alzheimer’s disease: synaptic transmission, plaques and the role of microglia

Brain region–specific lipid alterations in the PLB4 hBACE1 knock-in mouse model of Alzheimer’s disease

Knock, Knock, Knockin’ on Tranformativeness’ Doors

Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models

Age-related changes in brain phospholipids and bioactive lipids in the APP knock-in mouse model of Alzheimer’s disease

Microglial-associated responses to comorbid amyloid pathology and hyperhomocysteinemia in an aged knock-in mouse model of Alzheimer’s disease

Impact of hyperhomocysteinemia and different dietary interventions on cognitive performance in a knock-in mouse model for Alzheimer’s disease

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Impairment in novelty-promoted memory via behavioral tagging and capture before apparent memory loss in a knock-in model of Alzheimer’s disease

Vascular and parenchymal amyloid pathology in an Alzheimer disease knock-in mouse model: interplay with cerebral blood flow

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