Generation of App knock-in mice reveals deletion mutations protective against Alzheimer’s disease-like pathology

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2041-1723

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Generation of App knock-in mice reveals deletion mutations protective against Alzheimer’s disease-like pathology ; volume:9 ; number:1 ; day:4 ; month:5 ; year:2018 ; pages:1-7 ; date:12.2018
Nature Communications ; 9, Heft 1 (4.5.2018), 1-7, 12.2018

Creator: Nagata, Kenichi

Contributor: Takahashi, Mika
Matsuba, Yukio
Okuyama-Uchimura, Fumi
Sato, Kaori
Hashimoto, Shoko
Saito, Takashi
Saido, Takaomi C.
SpringerLink (Online service)

DOI: 10.1038/s41467-018-04238-0

URN: urn:nbn:de:101:1-2018070223181408180862

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:21 AM CEST

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Nagata, Kenichi
Takahashi, Mika
Matsuba, Yukio
Okuyama-Uchimura, Fumi
Sato, Kaori
Hashimoto, Shoko
Saito, Takashi
Saido, Takaomi C.
SpringerLink (Online service)

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Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models

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Age-related changes in brain phospholipids and bioactive lipids in the APP knock-in mouse model of Alzheimer’s disease

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Generation of App knock-in mice reveals deletion mutations protective against Alzheimer’s disease-like pathology

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Other Objects (12)

Microglial-associated responses to comorbid amyloid pathology and hyperhomocysteinemia in an aged knock-in mouse model of Alzheimer’s disease

Neuronal transcriptome, tau and synapse loss in Alzheimer’s knock-in mice require prion protein

Vascular and parenchymal amyloid pathology in an Alzheimer disease knock-in mouse model: interplay with cerebral blood flow

Knock-in models related to Alzheimer’s disease: synaptic transmission, plaques and the role of microglia

Primate-specific 82-kDa choline acetyltransferase attenuates progression of Alzheimer’s disease-like pathology in the APPNL-G-F knock-in mouse model

Protective effect of PDE4B subtype-specific inhibition in an App knock-in mouse model for Alzheimer’s disease

Brain region–specific lipid alterations in the PLB4 hBACE1 knock-in mouse model of Alzheimer’s disease

Knock, Knock, Knockin’ on Tranformativeness’ Doors

Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models

Impact of hyperhomocysteinemia and different dietary interventions on cognitive performance in a knock-in mouse model for Alzheimer’s disease

Age-related changes in brain phospholipids and bioactive lipids in the APP knock-in mouse model of Alzheimer’s disease

TDP-43 Pathology in Alzheimer’s Disease

Microglial-associated responses to comorbid amyloid pathology and hyperhomocysteinemia in an aged knock-in mouse model of Alzheimer’s disease

Neuronal transcriptome, tau and synapse loss in Alzheimer’s knock-in mice require prion protein

Vascular and parenchymal amyloid pathology in an Alzheimer disease knock-in mouse model: interplay with cerebral blood flow

Knock-in models related to Alzheimer’s disease: synaptic transmission, plaques and the role of microglia

Primate-specific 82-kDa choline acetyltransferase attenuates progression of Alzheimer’s disease-like pathology in the APPNL-G-F knock-in mouse model

Protective effect of PDE4B subtype-specific inhibition in an App knock-in mouse model for Alzheimer’s disease

Brain region–specific lipid alterations in the PLB4 hBACE1 knock-in mouse model of Alzheimer’s disease

Knock, Knock, Knockin’ on Tranformativeness’ Doors

Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models

Impact of hyperhomocysteinemia and different dietary interventions on cognitive performance in a knock-in mouse model for Alzheimer’s disease

Age-related changes in brain phospholipids and bioactive lipids in the APP knock-in mouse model of Alzheimer’s disease

TDP-43 Pathology in Alzheimer’s Disease

Microglial-associated responses to comorbid amyloid pathology and hyperhomocysteinemia in an aged knock-in mouse model of Alzheimer’s disease

Neuronal transcriptome, tau and synapse loss in Alzheimer’s knock-in mice require prion protein

Vascular and parenchymal amyloid pathology in an Alzheimer disease knock-in mouse model: interplay with cerebral blood flow

Knock-in models related to Alzheimer’s disease: synaptic transmission, plaques and the role of microglia

Primate-specific 82-kDa choline acetyltransferase attenuates progression of Alzheimer’s disease-like pathology in the APPNL-G-F knock-in mouse model

Protective effect of PDE4B subtype-specific inhibition in an App knock-in mouse model for Alzheimer’s disease

Brain region–specific lipid alterations in the PLB4 hBACE1 knock-in mouse model of Alzheimer’s disease

Knock, Knock, Knockin’ on Tranformativeness’ Doors

Mitochondrial hypermetabolism precedes impaired autophagy and synaptic disorganization in App knock-in Alzheimer mouse models

Impact of hyperhomocysteinemia and different dietary interventions on cognitive performance in a knock-in mouse model for Alzheimer’s disease

Age-related changes in brain phospholipids and bioactive lipids in the APP knock-in mouse model of Alzheimer’s disease

TDP-43 Pathology in Alzheimer’s Disease

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