β1-adrenoceptor stimulation promotes LPS-induced cardiomyocyte apoptosis through activating PKA and enhancing CaMKII and IκBα phosphorylation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1364-8535
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
β1-adrenoceptor stimulation promotes LPS-induced cardiomyocyte apoptosis through activating PKA and enhancing CaMKII and IκBα phosphorylation ; volume:19 ; number:1 ; day:1 ; month:12 ; year:2015 ; pages:1-15 ; date:12.2015
Critical care ; 19, Heft 1 (1.12.2015), 1-15, 12.2015
- Creator
-
Wang, Yiyang
Wang, Yuan
Yang, Duomeng
Yu, Xiaohui
Li, Hongmei
Lv, Xiuxiu
Lu, Daxiang
Wang, Huadong
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1186/s13054-015-0820-1
- URN
-
urn:nbn:de:101:1-2021102919493282204320
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:21 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wang, Yiyang
- Wang, Yuan
- Yang, Duomeng
- Yu, Xiaohui
- Li, Hongmei
- Lv, Xiuxiu
- Lu, Daxiang
- Wang, Huadong
- SpringerLink (Online service)
Other Objects (12)
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Trans-cinnamaldehyde protects against phenylephrine-induced cardiomyocyte hypertrophy through the CaMKII/ERK pathway
CaMKII and PKA-dependent phosphorylation co-regulate nuclear localization of HDAC4 in adult cardiomyocytes
Cardiomyocyte Na+ and Ca2+ mishandling drives vicious cycle involving CaMKII, ROS, and ryanodine receptors
Proteomic analysis of neonatal mouse hearts shows PKA functions as a cardiomyocyte replication regulator
Gemeindekarte S.O.VIII.16.; M.K.II.
Gemeindekarte S.O.VIII.16; M.K.II.
Alprostadil attenuates LPS-induced cardiomyocyte injury by inhibiting the Wnt5a/JNK/NF-κB pathway
Angiotensin-(1-9) prevents cardiomyocyte hypertrophy via miR-129-3p/PKIA/PKA signaling pathway
α-Adrenoceptor-vermittelte [Alpha-Adrenoceptor-vermittelte] Signaltransformation in menschlichen Thrombozyten
Decreased autophagy induced by β1-adrenoceptor autoantibodies contributes to cardiomyocyte apoptosis
N-Docosahexaenoylethanolamine ameliorates LPS-induced neuroinflammation via cAMP/PKA-dependent signaling
Atherogenic L5 LDL induces cardiomyocyte apoptosis and inhibits KATP channels through CaMKII activation
Trans-cinnamaldehyde protects against phenylephrine-induced cardiomyocyte hypertrophy through the CaMKII/ERK pathway
CaMKII and PKA-dependent phosphorylation co-regulate nuclear localization of HDAC4 in adult cardiomyocytes
Cardiomyocyte Na+ and Ca2+ mishandling drives vicious cycle involving CaMKII, ROS, and ryanodine receptors
Proteomic analysis of neonatal mouse hearts shows PKA functions as a cardiomyocyte replication regulator
Gemeindekarte S.O.VIII.16.; M.K.II.
Gemeindekarte S.O.VIII.16; M.K.II.
Alprostadil attenuates LPS-induced cardiomyocyte injury by inhibiting the Wnt5a/JNK/NF-κB pathway
Angiotensin-(1-9) prevents cardiomyocyte hypertrophy via miR-129-3p/PKIA/PKA signaling pathway
α-Adrenoceptor-vermittelte [Alpha-Adrenoceptor-vermittelte] Signaltransformation in menschlichen Thrombozyten
Decreased autophagy induced by β1-adrenoceptor autoantibodies contributes to cardiomyocyte apoptosis
N-Docosahexaenoylethanolamine ameliorates LPS-induced neuroinflammation via cAMP/PKA-dependent signaling
Atherogenic L5 LDL induces cardiomyocyte apoptosis and inhibits KATP channels through CaMKII activation
Trans-cinnamaldehyde protects against phenylephrine-induced cardiomyocyte hypertrophy through the CaMKII/ERK pathway
CaMKII and PKA-dependent phosphorylation co-regulate nuclear localization of HDAC4 in adult cardiomyocytes
Cardiomyocyte Na+ and Ca2+ mishandling drives vicious cycle involving CaMKII, ROS, and ryanodine receptors
Proteomic analysis of neonatal mouse hearts shows PKA functions as a cardiomyocyte replication regulator
Gemeindekarte S.O.VIII.16.; M.K.II.
Gemeindekarte S.O.VIII.16; M.K.II.
Alprostadil attenuates LPS-induced cardiomyocyte injury by inhibiting the Wnt5a/JNK/NF-κB pathway
Angiotensin-(1-9) prevents cardiomyocyte hypertrophy via miR-129-3p/PKIA/PKA signaling pathway