Activation of the unfolded protein response in high glucose treated endothelial cells is mediated by methylglyoxal
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-2322
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Activation of the unfolded protein response in high glucose treated endothelial cells is mediated by methylglyoxal ; volume:9 ; number:1 ; day:27 ; month:5 ; year:2019 ; pages:1-14 ; date:12.2019
Scientific reports ; 9, Heft 1 (27.5.2019), 1-14, 12.2019
- Classification
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Biowissenschaften, Biologie
- Creator
-
Irshad, Zehra
Xue, Mingzhan
Ashour, Amal
Larkin, James R.
Thornalley, Paul J.
Rabbani, Naila
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41598-019-44358-1
- URN
-
urn:nbn:de:101:1-2019101914233325784776
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:29 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Irshad, Zehra
- Xue, Mingzhan
- Ashour, Amal
- Larkin, James R.
- Thornalley, Paul J.
- Rabbani, Naila
- SpringerLink (Online service)
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Methylglyoxal induced endothelial dysfunction via loss of proliferation
Pathophysiology of methylglyoxal-associated endothelial damage in experimental sepsis
Role of glucose and reactive glucose metabolite methylglyoxal in pathophysiological blood vessel development in zebrafish
Glucose metabolite methylglyoxal induces vascular endothelial cell pyroptosis via NLRP3 inflammasome activation and oxidative stress in vitro and in vivo
Investigating the Glycating Effects of Glucose, Glyoxal and Methylglyoxal on Human Sperm
Endothelial cell and macrophage responses induced by methylglyoxal : potential role of mTORC1
Histidine containing dipeptides protect epithelial and endothelial cell barriers from methylglyoxal induced injury
Endothelial Na+/H+ exchanger NHE1 participates in redox-sensitive leukocyte recruitment triggered by methylglyoxal
Analysis of long non-coding RNA expression profiles in high-glucose treated vascular endothelial cells
High glucose mediates endothelial-to-chondrocyte transition in human aortic endothelial cells
MRPS6 modulates glucose-stimulated insulin secretion in mouse islet cells through mitochondrial unfolded protein response
Exosomal Mir-3613-3p derived from oxygen–glucose deprivation-treated brain microvascular endothelial cell promotes microglial M1 polarization
Methylglyoxal induced endothelial dysfunction via loss of proliferation
Pathophysiology of methylglyoxal-associated endothelial damage in experimental sepsis
Role of glucose and reactive glucose metabolite methylglyoxal in pathophysiological blood vessel development in zebrafish
Glucose metabolite methylglyoxal induces vascular endothelial cell pyroptosis via NLRP3 inflammasome activation and oxidative stress in vitro and in vivo
Investigating the Glycating Effects of Glucose, Glyoxal and Methylglyoxal on Human Sperm
Endothelial cell and macrophage responses induced by methylglyoxal : potential role of mTORC1
Histidine containing dipeptides protect epithelial and endothelial cell barriers from methylglyoxal induced injury
Endothelial Na+/H+ exchanger NHE1 participates in redox-sensitive leukocyte recruitment triggered by methylglyoxal
Analysis of long non-coding RNA expression profiles in high-glucose treated vascular endothelial cells
High glucose mediates endothelial-to-chondrocyte transition in human aortic endothelial cells
MRPS6 modulates glucose-stimulated insulin secretion in mouse islet cells through mitochondrial unfolded protein response