Context dependent isoform specific PI3K inhibition confers drug resistance in hepatocellular carcinoma cells

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1471-2407

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Context dependent isoform specific PI3K inhibition confers drug resistance in hepatocellular carcinoma cells ; volume:22 ; number:1 ; day:24 ; month:3 ; year:2022 ; pages:1-17 ; date:12.2022
BMC cancer ; 22, Heft 1 (24.3.2022), 1-17, 12.2022

Creator: Narci, Kubra
Kahraman, Deniz Cansen
Koyas, Altay
Ersahin, Tulin
Tuncbag, Nurcan
Atalay, Rengul Cetin

Contributor: SpringerLink (Online service)

DOI: 10.1186/s12885-022-09357-y

URN: urn:nbn:de:101:1-2022061522311671720656

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:32 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Narci, Kubra
Kahraman, Deniz Cansen
Koyas, Altay
Ersahin, Tulin
Tuncbag, Nurcan
Atalay, Rengul Cetin
SpringerLink (Online service)

Other Objects (12)

IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer

Activation of IGF1R/p110β/AKT/mTOR confers resistance to α-specific PI3K inhibition

EGFR signaling confers resistance to BET inhibition in hepatocellular carcinoma through stabilizing oncogenic MYC

PI3K-Inhibition im Pankreaskarzinommodell

EGFR blockade confers sensitivity to cabozantinib in hepatocellular carcinoma

PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition

A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells

PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Wnt signaling inhibition confers induced synthetic lethality to PARP inhibitors

Tollip promotes hepatocellular carcinoma progression via PI3K/AKT pathway

Correction: PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Dual mTOR/PI3K inhibition limits PI3K-dependent pathways activated upon mTOR inhibition in autosomal dominant polycystic kidney disease

IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer

Activation of IGF1R/p110β/AKT/mTOR confers resistance to α-specific PI3K inhibition

EGFR signaling confers resistance to BET inhibition in hepatocellular carcinoma through stabilizing oncogenic MYC

PI3K-Inhibition im Pankreaskarzinommodell

EGFR blockade confers sensitivity to cabozantinib in hepatocellular carcinoma

PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition

A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells

PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Wnt signaling inhibition confers induced synthetic lethality to PARP inhibitors

Tollip promotes hepatocellular carcinoma progression via PI3K/AKT pathway

Correction: PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Dual mTOR/PI3K inhibition limits PI3K-dependent pathways activated upon mTOR inhibition in autosomal dominant polycystic kidney disease

IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer

Activation of IGF1R/p110β/AKT/mTOR confers resistance to α-specific PI3K inhibition

EGFR signaling confers resistance to BET inhibition in hepatocellular carcinoma through stabilizing oncogenic MYC

PI3K-Inhibition im Pankreaskarzinommodell

EGFR blockade confers sensitivity to cabozantinib in hepatocellular carcinoma

PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition

A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells

PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Wnt signaling inhibition confers induced synthetic lethality to PARP inhibitors

Tollip promotes hepatocellular carcinoma progression via PI3K/AKT pathway

Correction: PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Dual mTOR/PI3K inhibition limits PI3K-dependent pathways activated upon mTOR inhibition in autosomal dominant polycystic kidney disease

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

Context dependent isoform specific PI3K inhibition confers drug resistance in hepatocellular carcinoma cells

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer

Activation of IGF1R/p110β/AKT/mTOR confers resistance to α-specific PI3K inhibition

EGFR signaling confers resistance to BET inhibition in hepatocellular carcinoma through stabilizing oncogenic MYC

PI3K-Inhibition im Pankreaskarzinommodell

EGFR blockade confers sensitivity to cabozantinib in hepatocellular carcinoma

PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition

A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells

PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Wnt signaling inhibition confers induced synthetic lethality to PARP inhibitors

Tollip promotes hepatocellular carcinoma progression via PI3K/AKT pathway

Correction: PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Dual mTOR/PI3K inhibition limits PI3K-dependent pathways activated upon mTOR inhibition in autosomal dominant polycystic kidney disease

IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer

Activation of IGF1R/p110β/AKT/mTOR confers resistance to α-specific PI3K inhibition

EGFR signaling confers resistance to BET inhibition in hepatocellular carcinoma through stabilizing oncogenic MYC

PI3K-Inhibition im Pankreaskarzinommodell

EGFR blockade confers sensitivity to cabozantinib in hepatocellular carcinoma

PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition

A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells

PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Wnt signaling inhibition confers induced synthetic lethality to PARP inhibitors

Tollip promotes hepatocellular carcinoma progression via PI3K/AKT pathway

Correction: PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Dual mTOR/PI3K inhibition limits PI3K-dependent pathways activated upon mTOR inhibition in autosomal dominant polycystic kidney disease

IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer

Activation of IGF1R/p110β/AKT/mTOR confers resistance to α-specific PI3K inhibition

EGFR signaling confers resistance to BET inhibition in hepatocellular carcinoma through stabilizing oncogenic MYC

PI3K-Inhibition im Pankreaskarzinommodell

EGFR blockade confers sensitivity to cabozantinib in hepatocellular carcinoma

PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition

A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells

PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Wnt signaling inhibition confers induced synthetic lethality to PARP inhibitors

Tollip promotes hepatocellular carcinoma progression via PI3K/AKT pathway

Correction: PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression

Dual mTOR/PI3K inhibition limits PI3K-dependent pathways activated upon mTOR inhibition in autosomal dominant polycystic kidney disease

Related objects

Reset password