Context dependent isoform specific PI3K inhibition confers drug resistance in hepatocellular carcinoma cells
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1471-2407
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Context dependent isoform specific PI3K inhibition confers drug resistance in hepatocellular carcinoma cells ; volume:22 ; number:1 ; day:24 ; month:3 ; year:2022 ; pages:1-17 ; date:12.2022
BMC cancer ; 22, Heft 1 (24.3.2022), 1-17, 12.2022
- Urheber
-
Narci, Kubra
Kahraman, Deniz Cansen
Koyas, Altay
Ersahin, Tulin
Tuncbag, Nurcan
Atalay, Rengul Cetin
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s12885-022-09357-y
- URN
-
urn:nbn:de:101:1-2022061522311671720656
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:32 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Narci, Kubra
- Kahraman, Deniz Cansen
- Koyas, Altay
- Ersahin, Tulin
- Tuncbag, Nurcan
- Atalay, Rengul Cetin
- SpringerLink (Online service)
Ähnliche Objekte (12)
IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer
Activation of IGF1R/p110β/AKT/mTOR confers resistance to α-specific PI3K inhibition
EGFR signaling confers resistance to BET inhibition in hepatocellular carcinoma through stabilizing oncogenic MYC
PI3K-Inhibition im Pankreaskarzinommodell
EGFR blockade confers sensitivity to cabozantinib in hepatocellular carcinoma
PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition
A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells
PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression
Wnt signaling inhibition confers induced synthetic lethality to PARP inhibitors
Tollip promotes hepatocellular carcinoma progression via PI3K/AKT pathway
Correction: PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression
Dual mTOR/PI3K inhibition limits PI3K-dependent pathways activated upon mTOR inhibition in autosomal dominant polycystic kidney disease
IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer
Activation of IGF1R/p110β/AKT/mTOR confers resistance to α-specific PI3K inhibition
EGFR signaling confers resistance to BET inhibition in hepatocellular carcinoma through stabilizing oncogenic MYC
PI3K-Inhibition im Pankreaskarzinommodell
EGFR blockade confers sensitivity to cabozantinib in hepatocellular carcinoma
PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition
A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells
PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression
Wnt signaling inhibition confers induced synthetic lethality to PARP inhibitors
Tollip promotes hepatocellular carcinoma progression via PI3K/AKT pathway
Correction: PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression
Dual mTOR/PI3K inhibition limits PI3K-dependent pathways activated upon mTOR inhibition in autosomal dominant polycystic kidney disease
IGF1R upregulation confers resistance to isoform-specific inhibitors of PI3K in PIK3CA-driven ovarian cancer
Activation of IGF1R/p110β/AKT/mTOR confers resistance to α-specific PI3K inhibition
EGFR signaling confers resistance to BET inhibition in hepatocellular carcinoma through stabilizing oncogenic MYC
PI3K-Inhibition im Pankreaskarzinommodell
EGFR blockade confers sensitivity to cabozantinib in hepatocellular carcinoma
PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition
A new ALK isoform transported by extracellular vesicles confers drug resistance to melanoma cells
PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression
Wnt signaling inhibition confers induced synthetic lethality to PARP inhibitors
Tollip promotes hepatocellular carcinoma progression via PI3K/AKT pathway
Correction: PTEN-regulated PI3K-p110 and AKT isoform plasticity controls metastatic prostate cancer progression