Impaired Ca2+ release contributes to muscle weakness in a rat model of critical illness myopathy

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1364-8535

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Impaired Ca2+ release contributes to muscle weakness in a rat model of critical illness myopathy ; volume:20 ; number:1 ; day:10 ; month:8 ; year:2016 ; pages:1-8 ; date:12.2016
Critical care ; 20, Heft 1 (10.8.2016), 1-8, 12.2016

Creator: Llano-Diez, Monica

Contributor: Cheng, Arthur J.
Jonsson, William
Ivarsson, Niklas
Westerblad, Håkan
Sun, Vic
Cacciani, Nicola
Larsson, Lars
Bruton, Joseph
SpringerLink (Online service)

DOI: 10.1186/s13054-016-1417-z

URN: urn:nbn:de:1111-201609086092

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:58 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Llano-Diez, Monica
Cheng, Arthur J.
Jonsson, William
Ivarsson, Niklas
Westerblad, Håkan
Sun, Vic
Cacciani, Nicola
Larsson, Lars
Bruton, Joseph
SpringerLink (Online service)

Other Objects (12)

Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long-term outcomes

Adolescent idiopathic scoliosis without limb weakness: a differential diagnosis of core myopathy?

A coordinated multiorgan metabolic response contributes to human mitochondrial myopathy

Publisher Correction to: Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long‑term outcomes

Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy

Muscle-tendon weakness contributes to chronic fatigue syndrome in Gaucher’s disease

ZBP1 contributes to interferonopathies caused by impaired ADAR1 function

Impaired L-type Ca2+ channel function in the dystrophic heart

Deletion of the Ste20-like kinase SLK in skeletal muscle results in a progressive myopathy and muscle weakness

Interferon-gamma-impaired vascular barrier function contributes to IBD pathogenesis

Impaired GAPDH ‐induced mitophagy contributes to the pathology of Huntington's disease

Increased temporal stride variability contributes to impaired gait coordination after stroke

Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long-term outcomes

Adolescent idiopathic scoliosis without limb weakness: a differential diagnosis of core myopathy?

A coordinated multiorgan metabolic response contributes to human mitochondrial myopathy

Publisher Correction to: Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long‑term outcomes

Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy

Muscle-tendon weakness contributes to chronic fatigue syndrome in Gaucher’s disease

ZBP1 contributes to interferonopathies caused by impaired ADAR1 function

Impaired L-type Ca2+ channel function in the dystrophic heart

Deletion of the Ste20-like kinase SLK in skeletal muscle results in a progressive myopathy and muscle weakness

Interferon-gamma-impaired vascular barrier function contributes to IBD pathogenesis

Impaired GAPDH ‐induced mitophagy contributes to the pathology of Huntington's disease

Increased temporal stride variability contributes to impaired gait coordination after stroke

Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long-term outcomes

Adolescent idiopathic scoliosis without limb weakness: a differential diagnosis of core myopathy?

A coordinated multiorgan metabolic response contributes to human mitochondrial myopathy

Publisher Correction to: Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long‑term outcomes

Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy

Muscle-tendon weakness contributes to chronic fatigue syndrome in Gaucher’s disease

ZBP1 contributes to interferonopathies caused by impaired ADAR1 function

Impaired L-type Ca2+ channel function in the dystrophic heart

Deletion of the Ste20-like kinase SLK in skeletal muscle results in a progressive myopathy and muscle weakness

Interferon-gamma-impaired vascular barrier function contributes to IBD pathogenesis

Impaired GAPDH ‐induced mitophagy contributes to the pathology of Huntington's disease

Increased temporal stride variability contributes to impaired gait coordination after stroke

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

Impaired Ca2+ release contributes to muscle weakness in a rat model of critical illness myopathy

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long-term outcomes

Adolescent idiopathic scoliosis without limb weakness: a differential diagnosis of core myopathy?

A coordinated multiorgan metabolic response contributes to human mitochondrial myopathy

Publisher Correction to: Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long‑term outcomes

Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy

Muscle-tendon weakness contributes to chronic fatigue syndrome in Gaucher’s disease

ZBP1 contributes to interferonopathies caused by impaired ADAR1 function

Impaired L-type Ca2+ channel function in the dystrophic heart

Deletion of the Ste20-like kinase SLK in skeletal muscle results in a progressive myopathy and muscle weakness

Interferon-gamma-impaired vascular barrier function contributes to IBD pathogenesis

Impaired GAPDH ‐induced mitophagy contributes to the pathology of Huntington's disease

Increased temporal stride variability contributes to impaired gait coordination after stroke

Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long-term outcomes

Adolescent idiopathic scoliosis without limb weakness: a differential diagnosis of core myopathy?

A coordinated multiorgan metabolic response contributes to human mitochondrial myopathy

Publisher Correction to: Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long‑term outcomes

Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy

Muscle-tendon weakness contributes to chronic fatigue syndrome in Gaucher’s disease

ZBP1 contributes to interferonopathies caused by impaired ADAR1 function

Impaired L-type Ca2+ channel function in the dystrophic heart

Deletion of the Ste20-like kinase SLK in skeletal muscle results in a progressive myopathy and muscle weakness

Interferon-gamma-impaired vascular barrier function contributes to IBD pathogenesis

Impaired GAPDH ‐induced mitophagy contributes to the pathology of Huntington's disease

Increased temporal stride variability contributes to impaired gait coordination after stroke

Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long-term outcomes

Adolescent idiopathic scoliosis without limb weakness: a differential diagnosis of core myopathy?

A coordinated multiorgan metabolic response contributes to human mitochondrial myopathy

Publisher Correction to: Critical Illness Weakness, Polyneuropathy and Myopathy: Diagnosis, treatment, and long‑term outcomes

Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy

Muscle-tendon weakness contributes to chronic fatigue syndrome in Gaucher’s disease

ZBP1 contributes to interferonopathies caused by impaired ADAR1 function

Impaired L-type Ca2+ channel function in the dystrophic heart

Deletion of the Ste20-like kinase SLK in skeletal muscle results in a progressive myopathy and muscle weakness

Interferon-gamma-impaired vascular barrier function contributes to IBD pathogenesis

Impaired GAPDH ‐induced mitophagy contributes to the pathology of Huntington's disease

Increased temporal stride variability contributes to impaired gait coordination after stroke

Related objects

Reset password