Impaired GAPDH ‐induced mitophagy contributes to the pathology of Huntington's disease

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: Impaired GAPDH ‐induced mitophagy contributes to the pathology of Huntington's disease ; volume:7 ; number:10 ; year:2015 ; pages:1307-1326 ; extent:20
EMBO molecular medicine / European Molecular Biology Organization ; 7, Heft 10 (2015), 1307-1326 (gesamt 20)

Creator: Hwang, Sunhee
Disatnik, Marie‐Hélène
Mochly‐Rosen, Daria

DOI: 10.15252/emmm.201505256

URN: urn:nbn:de:101:1-2022120406151303522301

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:36 AM CEST

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Hwang, Sunhee
Disatnik, Marie‐Hélène
Mochly‐Rosen, Daria

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ZIP7 contributes to the pathogenesis of diabetic cardiomyopathy by suppressing mitophagy in mouse hearts

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Impaired GAPDH ‐induced mitophagy contributes to the pathology of Huntington's disease

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Other Objects (12)

Impaired mitophagy induces antimicrobial responses in macrophages infected with Mycobacterium tuberculosis

Mitochondrial damage and impaired mitophagy contribute to disease progression in SCA6

USP 14 inhibition corrects an in vivo model of impaired mitophagy

Altered m6A RNA methylation contributes to hippocampal memory deficits in Huntington’s disease mice

Pancreatic β cells overexpressing hIAPP impaired mitophagy and unbalanced mitochondrial dynamics

Nebulization of low-dose aspirin ameliorates Huntington’s pathology in N171-82Q transgenic mice

Manifestation of Huntington’s disease pathology in human induced pluripotent stem cell-derived neurons

Neuron type‐specific increase in lamin B1 contributes to nuclear dysfunction in Huntington’s disease

ULK1-mediated phosphorylation of ATG14 promotes autophagy and is impaired in Huntington’s disease models

ZBP1 contributes to interferonopathies caused by impaired ADAR1 function

ZIP7 contributes to the pathogenesis of diabetic cardiomyopathy by suppressing mitophagy in mouse hearts

Thalamostriatal degeneration contributes to dystonia and cholinergic interneuron dysfunction in a mouse model of Huntington’s disease

Impaired mitophagy induces antimicrobial responses in macrophages infected with Mycobacterium tuberculosis

Mitochondrial damage and impaired mitophagy contribute to disease progression in SCA6

USP 14 inhibition corrects an in vivo model of impaired mitophagy

Altered m6A RNA methylation contributes to hippocampal memory deficits in Huntington’s disease mice

Pancreatic β cells overexpressing hIAPP impaired mitophagy and unbalanced mitochondrial dynamics

Nebulization of low-dose aspirin ameliorates Huntington’s pathology in N171-82Q transgenic mice

Manifestation of Huntington’s disease pathology in human induced pluripotent stem cell-derived neurons

Neuron type‐specific increase in lamin B1 contributes to nuclear dysfunction in Huntington’s disease

ULK1-mediated phosphorylation of ATG14 promotes autophagy and is impaired in Huntington’s disease models

ZBP1 contributes to interferonopathies caused by impaired ADAR1 function

ZIP7 contributes to the pathogenesis of diabetic cardiomyopathy by suppressing mitophagy in mouse hearts

Thalamostriatal degeneration contributes to dystonia and cholinergic interneuron dysfunction in a mouse model of Huntington’s disease

Impaired mitophagy induces antimicrobial responses in macrophages infected with Mycobacterium tuberculosis

Mitochondrial damage and impaired mitophagy contribute to disease progression in SCA6

USP 14 inhibition corrects an in vivo model of impaired mitophagy

Altered m6A RNA methylation contributes to hippocampal memory deficits in Huntington’s disease mice

Pancreatic β cells overexpressing hIAPP impaired mitophagy and unbalanced mitochondrial dynamics

Nebulization of low-dose aspirin ameliorates Huntington’s pathology in N171-82Q transgenic mice

Manifestation of Huntington’s disease pathology in human induced pluripotent stem cell-derived neurons

Neuron type‐specific increase in lamin B1 contributes to nuclear dysfunction in Huntington’s disease

ULK1-mediated phosphorylation of ATG14 promotes autophagy and is impaired in Huntington’s disease models

ZBP1 contributes to interferonopathies caused by impaired ADAR1 function

ZIP7 contributes to the pathogenesis of diabetic cardiomyopathy by suppressing mitophagy in mouse hearts

Thalamostriatal degeneration contributes to dystonia and cholinergic interneuron dysfunction in a mouse model of Huntington’s disease

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