Elucidating the Role of Cerebellar Synaptic Dysfunction in C9orf72-ALS/FTD — a Systematic Review and Meta-Analysis
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1473-4230
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Elucidating the Role of Cerebellar Synaptic Dysfunction in C9orf72-ALS/FTD — a Systematic Review and Meta-Analysis ; day:7 ; month:9 ; year:2021 ; pages:1-34
The cerebellum ; (7.9.2021), 1-34
- Urheber
-
Kaliszewska, Aleksandra
Allison, Joseph
Col, Tarik-Tarkan
Shaw, Christopher
Arias, Natalia
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1007/s12311-021-01320-0
- URN
-
urn:nbn:de:101:1-2021111918043108658165
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:33 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Kaliszewska, Aleksandra
- Allison, Joseph
- Col, Tarik-Tarkan
- Shaw, Christopher
- Arias, Natalia
- SpringerLink (Online service)
Ähnliche Objekte (12)
Molecular pathology, developmental changes and synaptic dysfunction in (pre-) symptomatic human C9ORF72-ALS/FTD cerebral organoids
Synaptic dysfunction induced by glycine‐alanine dipeptides in C9orf72‐ ALS/FTD is rescued by SV 2 replenishment
A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma
Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit
Nuclear lamina invaginations are not a pathological feature of C9orf72 ALS/FTD
A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD
RNA dependent suppression of C9orf72 ALS/FTD associated neurodegeneration by Matrin-3
Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD
Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell
Synaptic localization of C9orf72 regulates post-synaptic glutamate receptor 1 levels
Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD
Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies
Molecular pathology, developmental changes and synaptic dysfunction in (pre-) symptomatic human C9ORF72-ALS/FTD cerebral organoids
Synaptic dysfunction induced by glycine‐alanine dipeptides in C9orf72‐ ALS/FTD is rescued by SV 2 replenishment
A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma
Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit
Nuclear lamina invaginations are not a pathological feature of C9orf72 ALS/FTD
A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD
RNA dependent suppression of C9orf72 ALS/FTD associated neurodegeneration by Matrin-3
Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD
Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell
Synaptic localization of C9orf72 regulates post-synaptic glutamate receptor 1 levels
Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD
Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies
Molecular pathology, developmental changes and synaptic dysfunction in (pre-) symptomatic human C9ORF72-ALS/FTD cerebral organoids
Synaptic dysfunction induced by glycine‐alanine dipeptides in C9orf72‐ ALS/FTD is rescued by SV 2 replenishment
A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma
Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit
Nuclear lamina invaginations are not a pathological feature of C9orf72 ALS/FTD
A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD
RNA dependent suppression of C9orf72 ALS/FTD associated neurodegeneration by Matrin-3
Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD
Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell
Synaptic localization of C9orf72 regulates post-synaptic glutamate receptor 1 levels
Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD