ESAM supports neutrophil extravasation, activation of Rho and VEGF-induced vascular permeability
- Location
-
München, Bayerische Staatsbibliothek -- 4 Z 2001.231-203,5/8#S.1671-1677
- Extent
-
1 Online-Ressource (S. 1671 - 1677)
- Language
-
Englisch
- Notes
-
graph. Darst.
In: The journal of experimental medicine, Vol. 203, No. 7, 2006. - New York, NY : Rockefeller Univ. Press
- Event
-
Veröffentlichung
- (where)
-
New York, NY
- (who)
-
Rockefeller Univ. Press
- (when)
-
2006
- Contributor
- URN
-
urn:nbn:de:bvb:12-bsb00060030-0
- Last update
-
16.04.2025, 8:18 AM CEST
Data provider
This object is provided by:
Bayerische Staatsbibliothek. If you have any questions about the object, please contact the data provider.
Bayerische Staatsbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wegmann, Frank
- Vestweber, Dietmar
- Rockefeller Univ. Press
Time of origin
- 2006
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![[Rho]](https://iiif.deutsche-digitale-bibliothek.de/image/2/96d9d4bf-761d-49e1-9cd1-70f0c12992d4/full/!306,450/0/default.jpg)
[Rho]
RHO
Endothelial molecules in the process of leukocyte extravasation
1-Palmitoyl-2-linoleoyl-3-acetyl-rac-glycerol (PLAG) attenuates gemcitabine-induced neutrophil extravasation
A CD99-related antigen on endothelial cells mediates neutrophil but not lymphocyte extravasation in vivo
LFA-1 (CD11a/CD18) and Mac-1 (CD11b/CD18) distinctly regulate neutrophil extravasation through hotspots I and II
Vasculotide reduces endothelial permeability and tumor cell extravasation in the absence of binding to or agonistic activation of Tie2
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Delayed emergence due to remimazolam extravasation
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R-spondin 2 mediates neutrophil egress into the alveolar space through increased lung permeability
[Rho]
RHO
Endothelial molecules in the process of leukocyte extravasation
1-Palmitoyl-2-linoleoyl-3-acetyl-rac-glycerol (PLAG) attenuates gemcitabine-induced neutrophil extravasation
A CD99-related antigen on endothelial cells mediates neutrophil but not lymphocyte extravasation in vivo
LFA-1 (CD11a/CD18) and Mac-1 (CD11b/CD18) distinctly regulate neutrophil extravasation through hotspots I and II
Vasculotide reduces endothelial permeability and tumor cell extravasation in the absence of binding to or agonistic activation of Tie2
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[Rho]
RHO