Slit2/Robo4 Signaling: Potential Role of a VEGF-Antagonist Pathway to Regulate Luteal Permeability

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Alternative title: Slit2/Robo4 Signaling: Potenzielle Rolle eines VEGF-antagonistischen Systems in der Regulation der lutealen Permeabilität

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: Slit2/Robo4 Signaling: Potential Role of a VEGF-Antagonist Pathway to Regulate Luteal Permeability ; volume:77 ; number:01 ; year:2017 ; pages:73-80
Geburtshilfe und Frauenheilkunde ; 77, Heft 01 (2017), 73-80

Contributor: Bekes, I.
Haunerdinger, V.
Sauter, R.
Holzheu, I.
Janni, W.
Wöckel, A.
Wulff, C.

DOI: 10.1055/s-0042-113461

URN: urn:nbn:de:101:1-2018100111371637516234

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:28 AM CEST

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Bekes, I.
Haunerdinger, V.
Sauter, R.
Holzheu, I.
Janni, W.
Wöckel, A.
Wulff, C.

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Slit2/Robo4 Signaling: Potential Role of a VEGF-Antagonist Pathway to Regulate Luteal Permeability

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Other Objects (12)

Slit2N and Robo4 regulate lymphangiogenesis through the VEGF-C/VEGFR-3 pathway

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Silver nanoparticles inhibit VEGF-and IL-1β-induced vascular permeability via Src dependent pathway in porcine retinal endothelial cells

ESAM supports neutrophil extravasation, activation of Rho and VEGF-induced vascular permeability

The Kynurenine Pathway in Gut Permeability and Inflammation

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Plasmodium falciparum sexual parasites regulate infected erythrocyte permeability

Pericytes regulate VEGF-induced endothelial sprouting through VEGFR1

VEGF and TNF up-regulate, NSAID down-regulate SOX18 protein level in HUVEC

Silver nanoparticles inhibit VEGF-and IL-1β-induced vascular permeability via Src dependent pathway in porcine retinal endothelial cells

ESAM supports neutrophil extravasation, activation of Rho and VEGF-induced vascular permeability

The Kynurenine Pathway in Gut Permeability and Inflammation

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YAP1 and the Hippo Signaling Pathway Regulate Progenitor Proliferation

Slit2N and Robo4 regulate lymphangiogenesis through the VEGF-C/VEGFR-3 pathway

Plasmodium falciparum sexual parasites regulate infected erythrocyte permeability

Pericytes regulate VEGF-induced endothelial sprouting through VEGFR1

VEGF and TNF up-regulate, NSAID down-regulate SOX18 protein level in HUVEC

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ESAM supports neutrophil extravasation, activation of Rho and VEGF-induced vascular permeability

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