Systematic Analysis of Molecular and Cellular Dysfunction in Accelerated Aging Phenotypes
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
Göttingen, Georg-August Universität, Dissertation, 2021
- Schlagwort
-
Aging
Phenotype
- Ereignis
-
Veröffentlichung
- (wo)
-
Göttingen
- (wer)
-
Niedersächsische Staats- und Universitätsbibliothek Göttingen
- (wann)
-
2021
- Urheber
- Handle
-
21.11130/00-1735-0000-0008-591A-8
- URN
-
urn:nbn:de:gbv:7-21.11130/00-1735-0000-0008-591A-8-0
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
24.08.20252025, 14:37 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Werner, Gesa
- Niedersächsische Staats- und Universitätsbibliothek Göttingen
Entstanden
- 2021
Ähnliche Objekte (12)
Cellular aging is accelerated in the malignant clone of myeloproliferative neoplasms
Cellular aging is accelerated in the malignant clone of myeloproliferative neoplasms
New insights into the molecularpathogenesis of accelerated aging phenotypes in Hallermann-Streiff and Bloom syndromes
Down syndrome, accelerated aging and immunosenescence
Cellular models of aging
Mechanisms of aging in senescence-accelerated mice
Physiological aging and inflammation-induced cellular senescence may contribute to oligodendroglial dysfunction in MS
Loss of a single allele for Ku80 leads to progenitor dysfunction and accelerated aging in skeletal muscle
Redefining Aging in HIV Infection Using Phenotypes
Colour Stability of Veneering Composites after Accelerated Aging
Accelerated aging test modeling applied to solar mirrors
Microglia phenotypes converge in aging and neurodegenerative disease
Cellular aging is accelerated in the malignant clone of myeloproliferative neoplasms
Cellular aging is accelerated in the malignant clone of myeloproliferative neoplasms
New insights into the molecularpathogenesis of accelerated aging phenotypes in Hallermann-Streiff and Bloom syndromes
Down syndrome, accelerated aging and immunosenescence
Cellular models of aging
Mechanisms of aging in senescence-accelerated mice
Physiological aging and inflammation-induced cellular senescence may contribute to oligodendroglial dysfunction in MS
Loss of a single allele for Ku80 leads to progenitor dysfunction and accelerated aging in skeletal muscle
Redefining Aging in HIV Infection Using Phenotypes
Colour Stability of Veneering Composites after Accelerated Aging
Accelerated aging test modeling applied to solar mirrors
Microglia phenotypes converge in aging and neurodegenerative disease
Cellular aging is accelerated in the malignant clone of myeloproliferative neoplasms
Cellular aging is accelerated in the malignant clone of myeloproliferative neoplasms
New insights into the molecularpathogenesis of accelerated aging phenotypes in Hallermann-Streiff and Bloom syndromes
Down syndrome, accelerated aging and immunosenescence
Cellular models of aging
Mechanisms of aging in senescence-accelerated mice
Physiological aging and inflammation-induced cellular senescence may contribute to oligodendroglial dysfunction in MS
Loss of a single allele for Ku80 leads to progenitor dysfunction and accelerated aging in skeletal muscle
Redefining Aging in HIV Infection Using Phenotypes
Colour Stability of Veneering Composites after Accelerated Aging
Accelerated aging test modeling applied to solar mirrors