DNA hydroxymethylation is associated with disease severity and persists at enhancers of oncogenic regions in multiple myeloma
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1868-7083
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
DNA hydroxymethylation is associated with disease severity and persists at enhancers of oncogenic regions in multiple myeloma ; volume:12 ; number:1 ; day:2 ; month:11 ; year:2020 ; pages:1-12 ; date:12.2020
Clinical epigenetics ; 12, Heft 1 (2.11.2020), 1-12, 12.2020
- Urheber
-
Alberge, Jean-Baptiste
Magrangeas, Florence
Wagner, Mirko
Denié, Soline
Guérin-Charbonnel, Catherine
Campion, Loïc
Attal, Michel
Avet-Loiseau, Hervé
Carell, Thomas
Moreau, Philippe
Minvielle, Stéphane
Sérandour, Aurélien A.
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s13148-020-00953-y
- URN
-
urn:nbn:de:101:1-2020121520243466058922
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:36 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Alberge, Jean-Baptiste
- Magrangeas, Florence
- Wagner, Mirko
- Denié, Soline
- Guérin-Charbonnel, Catherine
- Campion, Loïc
- Attal, Michel
- Avet-Loiseau, Hervé
- Carell, Thomas
- Moreau, Philippe
- Minvielle, Stéphane
- Sérandour, Aurélien A.
- SpringerLink (Online service)
Ähnliche Objekte (12)
Emergence of enhancers at late DNA replicating regions
HPV16-miRNAs exert oncogenic effects through enhancers in human cervical cancer
SMARCB1 loss activates patient-specific distal oncogenic enhancers in malignant rhabdoid tumors
Cyclin K and cyclin D1b are oncogenic in myeloma cells
Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory
Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory
Polycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanisms
Enhancers, enhancers – from their discovery to today’s universe of transcription enhancers
Understanding super-enhancers
Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers
Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease
Whole-genome sequencing of multiple myeloma reveals oncogenic pathways are targeted somatically through multiple mechanisms
Emergence of enhancers at late DNA replicating regions
HPV16-miRNAs exert oncogenic effects through enhancers in human cervical cancer
SMARCB1 loss activates patient-specific distal oncogenic enhancers in malignant rhabdoid tumors
Cyclin K and cyclin D1b are oncogenic in myeloma cells
Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory
Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory
Polycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanisms
Enhancers, enhancers – from their discovery to today’s universe of transcription enhancers
Understanding super-enhancers
Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers
Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease
Whole-genome sequencing of multiple myeloma reveals oncogenic pathways are targeted somatically through multiple mechanisms
Emergence of enhancers at late DNA replicating regions
HPV16-miRNAs exert oncogenic effects through enhancers in human cervical cancer
SMARCB1 loss activates patient-specific distal oncogenic enhancers in malignant rhabdoid tumors
Cyclin K and cyclin D1b are oncogenic in myeloma cells
Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory
Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory
Polycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanisms
Enhancers, enhancers – from their discovery to today’s universe of transcription enhancers
Understanding super-enhancers
Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers
Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease