DNA hydroxymethylation is associated with disease severity and persists at enhancers of oncogenic regions in multiple myeloma

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1868-7083

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: DNA hydroxymethylation is associated with disease severity and persists at enhancers of oncogenic regions in multiple myeloma ; volume:12 ; number:1 ; day:2 ; month:11 ; year:2020 ; pages:1-12 ; date:12.2020
Clinical epigenetics ; 12, Heft 1 (2.11.2020), 1-12, 12.2020

Creator: Alberge, Jean-Baptiste
Magrangeas, Florence
Wagner, Mirko
Denié, Soline
Guérin-Charbonnel, Catherine
Campion, Loïc
Attal, Michel
Avet-Loiseau, Hervé
Carell, Thomas
Moreau, Philippe
Minvielle, Stéphane
Sérandour, Aurélien A.

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13148-020-00953-y

URN: urn:nbn:de:101:1-2020121520243466058922

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:36 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Alberge, Jean-Baptiste
Magrangeas, Florence
Wagner, Mirko
Denié, Soline
Guérin-Charbonnel, Catherine
Campion, Loïc
Attal, Michel
Avet-Loiseau, Hervé
Carell, Thomas
Moreau, Philippe
Minvielle, Stéphane
Sérandour, Aurélien A.
SpringerLink (Online service)

Other Objects (12)

Emergence of enhancers at late DNA replicating regions

HPV16-miRNAs exert oncogenic effects through enhancers in human cervical cancer

SMARCB1 loss activates patient-specific distal oncogenic enhancers in malignant rhabdoid tumors

Cyclin K and cyclin D1b are oncogenic in myeloma cells

Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory

Polycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanisms

Enhancers, enhancers – from their discovery to today’s universe of transcription enhancers

Understanding super-enhancers

Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers

$Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease$

Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease

Whole-genome sequencing of multiple myeloma reveals oncogenic pathways are targeted somatically through multiple mechanisms

Emergence of enhancers at late DNA replicating regions

HPV16-miRNAs exert oncogenic effects through enhancers in human cervical cancer

SMARCB1 loss activates patient-specific distal oncogenic enhancers in malignant rhabdoid tumors

Cyclin K and cyclin D1b are oncogenic in myeloma cells

Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory

Polycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanisms

Enhancers, enhancers – from their discovery to today’s universe of transcription enhancers

Understanding super-enhancers

Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers

$Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease$

Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease

Whole-genome sequencing of multiple myeloma reveals oncogenic pathways are targeted somatically through multiple mechanisms

Emergence of enhancers at late DNA replicating regions

HPV16-miRNAs exert oncogenic effects through enhancers in human cervical cancer

SMARCB1 loss activates patient-specific distal oncogenic enhancers in malignant rhabdoid tumors

Cyclin K and cyclin D1b are oncogenic in myeloma cells

Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory

Polycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanisms

Enhancers, enhancers – from their discovery to today’s universe of transcription enhancers

Understanding super-enhancers

Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers

$Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease$

Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease

Whole-genome sequencing of multiple myeloma reveals oncogenic pathways are targeted somatically through multiple mechanisms

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

DNA hydroxymethylation is associated with disease severity and persists at enhancers of oncogenic regions in multiple myeloma

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Emergence of enhancers at late DNA replicating regions

HPV16-miRNAs exert oncogenic effects through enhancers in human cervical cancer

SMARCB1 loss activates patient-specific distal oncogenic enhancers in malignant rhabdoid tumors

Cyclin K and cyclin D1b are oncogenic in myeloma cells

Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory

Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory

Polycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanisms

Enhancers, enhancers – from their discovery to today’s universe of transcription enhancers

Understanding super-enhancers

Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers

Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease

Whole-genome sequencing of multiple myeloma reveals oncogenic pathways are targeted somatically through multiple mechanisms

Emergence of enhancers at late DNA replicating regions

HPV16-miRNAs exert oncogenic effects through enhancers in human cervical cancer

SMARCB1 loss activates patient-specific distal oncogenic enhancers in malignant rhabdoid tumors

Cyclin K and cyclin D1b are oncogenic in myeloma cells

Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory

Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory

Polycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanisms

Enhancers, enhancers – from their discovery to today’s universe of transcription enhancers

Understanding super-enhancers

Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers

Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease

Whole-genome sequencing of multiple myeloma reveals oncogenic pathways are targeted somatically through multiple mechanisms

Emergence of enhancers at late DNA replicating regions

HPV16-miRNAs exert oncogenic effects through enhancers in human cervical cancer

SMARCB1 loss activates patient-specific distal oncogenic enhancers in malignant rhabdoid tumors

Cyclin K and cyclin D1b are oncogenic in myeloma cells

Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory

Oncogenic enhancers prime quiescent metastatic cells to escape NK immune surveillance by eliciting transcriptional memory

Polycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanisms

Enhancers, enhancers – from their discovery to today’s universe of transcription enhancers

Understanding super-enhancers

Differentially methylated regions within lung cancer risk loci are enriched in deregulated enhancers

Functional analysis of oncogenic lesions in multiple myeloma with potential significance for refractory disease

Whole-genome sequencing of multiple myeloma reveals oncogenic pathways are targeted somatically through multiple mechanisms

Related objects

Reset password