The lncRNA LAMP5-AS1 drives leukemia cell stemness by directly modulating DOT1L methyltransferase activity in MLL leukemia
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1756-8722
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
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The lncRNA LAMP5-AS1 drives leukemia cell stemness by directly modulating DOT1L methyltransferase activity in MLL leukemia ; volume:13 ; number:1 ; day:17 ; month:6 ; year:2020 ; pages:1-19 ; date:12.2020
Journal of hematology & oncology ; 13, Heft 1 (17.6.2020), 1-19, 12.2020
- Creator
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Wang, Wen-Tao
Chen, Tian-Qi
Zeng, Zhan-Cheng
Pan, Qi
Huang, Wei
Han, Cai
Fang, Ke
Sun, Lin-Yu
Yang, Qian-Qian
Wang, Dan
Luo, Xue-Qun
Sun, Yu-Meng
Chen, Yue-Qin
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s13045-020-00909-y
- URN
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urn:nbn:de:101:1-2020081411465995130585
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:54 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wang, Wen-Tao
- Chen, Tian-Qi
- Zeng, Zhan-Cheng
- Pan, Qi
- Huang, Wei
- Han, Cai
- Fang, Ke
- Sun, Lin-Yu
- Yang, Qian-Qian
- Wang, Dan
- Luo, Xue-Qun
- Sun, Yu-Meng
- Chen, Yue-Qin
- SpringerLink (Online service)
Other Objects (12)
Correction to: The lncRNA LAMP5-AS1 drives leukemia cell stemness by directly modulating DOT1L methyltransferase activity in MLL leukemia
MLL-r Leukemia
MLL oncoprotein levels influence leukemia lineage identities
RNA binding proteins in MLL-rearranged leukemia
How to effectively treat acute leukemia patients bearing MLL-rearrangements ?
MLL-fusion-driven leukemia requires SETD2 to safeguard genomic integrity
Pharmacological inhibition of LSD1 for the treatment of MLL-rearranged leukemia
SUV39H1 regulates the progression of MLL-AF9-induced acute myeloid leukemia
Thiopurine methyltransferase polymorphisms in children with acute lymphoblastic leukemia
FBXO22 promotes leukemogenesis by targeting BACH1 in MLL-rearranged acute myeloid leukemia
Functional diversity of inhibitors tackling the differentiation blockage of MLL-rearranged leukemia
Thiopurine methyltransferase genotyping in Palestinian childhood acute lymphoblastic leukemia patients
Correction to: The lncRNA LAMP5-AS1 drives leukemia cell stemness by directly modulating DOT1L methyltransferase activity in MLL leukemia
MLL-r Leukemia
MLL oncoprotein levels influence leukemia lineage identities
RNA binding proteins in MLL-rearranged leukemia
How to effectively treat acute leukemia patients bearing MLL-rearrangements ?
MLL-fusion-driven leukemia requires SETD2 to safeguard genomic integrity
Pharmacological inhibition of LSD1 for the treatment of MLL-rearranged leukemia
SUV39H1 regulates the progression of MLL-AF9-induced acute myeloid leukemia
Thiopurine methyltransferase polymorphisms in children with acute lymphoblastic leukemia
FBXO22 promotes leukemogenesis by targeting BACH1 in MLL-rearranged acute myeloid leukemia
Functional diversity of inhibitors tackling the differentiation blockage of MLL-rearranged leukemia
Thiopurine methyltransferase genotyping in Palestinian childhood acute lymphoblastic leukemia patients
Correction to: The lncRNA LAMP5-AS1 drives leukemia cell stemness by directly modulating DOT1L methyltransferase activity in MLL leukemia
MLL-r Leukemia
MLL oncoprotein levels influence leukemia lineage identities
RNA binding proteins in MLL-rearranged leukemia
How to effectively treat acute leukemia patients bearing MLL-rearrangements ?
MLL-fusion-driven leukemia requires SETD2 to safeguard genomic integrity
Pharmacological inhibition of LSD1 for the treatment of MLL-rearranged leukemia
SUV39H1 regulates the progression of MLL-AF9-induced acute myeloid leukemia
Thiopurine methyltransferase polymorphisms in children with acute lymphoblastic leukemia
FBXO22 promotes leukemogenesis by targeting BACH1 in MLL-rearranged acute myeloid leukemia
Functional diversity of inhibitors tackling the differentiation blockage of MLL-rearranged leukemia