- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
MLL oncoprotein levels influence leukemia lineage identities ; volume:15 ; number:1 ; day:29 ; month:10 ; year:2024 ; pages:1-13 ; date:12.2024
Nature Communications ; 15, Heft 1 (29.10.2024), 1-13, 12.2024
- Creator
-
Janssens, Derek H.
Duran, Melodie
Otto, Dominik J.
Wu, Weifang
Xu, Yiling
Kirkey, Danielle
Mullighan, Charles G.
Yi, Joanna S.
Meshinchi, Soheil
Sarthy, Jay F.
Ahmad, Kami
Henikoff, Steven
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41467-024-53399-8
- URN
-
urn:nbn:de:101:1-2501182111257.909217359048
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:20 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Janssens, Derek H.
- Duran, Melodie
- Otto, Dominik J.
- Wu, Weifang
- Xu, Yiling
- Kirkey, Danielle
- Mullighan, Charles G.
- Yi, Joanna S.
- Meshinchi, Soheil
- Sarthy, Jay F.
- Ahmad, Kami
- Henikoff, Steven
- SpringerLink (Online service)
Other Objects (12)
MLL-r Leukemia
MLL rearrangements in pediatric acute lymphoblastic and myeloblastic leukemias: MLL specific and lineage specific signatures
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How to effectively treat acute leukemia patients bearing MLL-rearrangements ?
MLL-fusion-driven leukemia requires SETD2 to safeguard genomic integrity
Pharmacological inhibition of LSD1 for the treatment of MLL-rearranged leukemia
SUV39H1 regulates the progression of MLL-AF9-induced acute myeloid leukemia
WTZ MLL
WTZ MLL
circRNA circAF4 functions as an oncogene to regulate MLL-AF4 fusion protein expression and inhibit MLL leukemia progression
FBXO22 promotes leukemogenesis by targeting BACH1 in MLL-rearranged acute myeloid leukemia
Functional diversity of inhibitors tackling the differentiation blockage of MLL-rearranged leukemia
MLL-r Leukemia
MLL rearrangements in pediatric acute lymphoblastic and myeloblastic leukemias: MLL specific and lineage specific signatures
RNA binding proteins in MLL-rearranged leukemia
How to effectively treat acute leukemia patients bearing MLL-rearrangements ?
MLL-fusion-driven leukemia requires SETD2 to safeguard genomic integrity
Pharmacological inhibition of LSD1 for the treatment of MLL-rearranged leukemia
SUV39H1 regulates the progression of MLL-AF9-induced acute myeloid leukemia
WTZ MLL
WTZ MLL
circRNA circAF4 functions as an oncogene to regulate MLL-AF4 fusion protein expression and inhibit MLL leukemia progression
FBXO22 promotes leukemogenesis by targeting BACH1 in MLL-rearranged acute myeloid leukemia
Functional diversity of inhibitors tackling the differentiation blockage of MLL-rearranged leukemia
MLL-r Leukemia
MLL rearrangements in pediatric acute lymphoblastic and myeloblastic leukemias: MLL specific and lineage specific signatures
RNA binding proteins in MLL-rearranged leukemia
How to effectively treat acute leukemia patients bearing MLL-rearrangements ?
MLL-fusion-driven leukemia requires SETD2 to safeguard genomic integrity
Pharmacological inhibition of LSD1 for the treatment of MLL-rearranged leukemia
SUV39H1 regulates the progression of MLL-AF9-induced acute myeloid leukemia
WTZ MLL
WTZ MLL
circRNA circAF4 functions as an oncogene to regulate MLL-AF4 fusion protein expression and inhibit MLL leukemia progression
FBXO22 promotes leukemogenesis by targeting BACH1 in MLL-rearranged acute myeloid leukemia