Multiallelic copy number variation in the complement component 4A (C4A) gene is associated with late-stage age-related macular degeneration (AMD)
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1742-2094
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Multiallelic copy number variation in the complement component 4A (C4A) gene is associated with late-stage age-related macular degeneration (AMD) ; volume:13 ; number:1 ; day:18 ; month:4 ; year:2016 ; pages:1-9 ; date:12.2016
Journal of neuroinflammation ; 13, Heft 1 (18.4.2016), 1-9, 12.2016
- Creator
-
Grassmann, Felix
- Contributor
-
Cantsilieris, Stuart
Schulz-Kuhnt, Anja-Sabrina
White, Stefan J.
Richardson, Andrea J.
Hewitt, Alex W.
Vote, Brendan J.
Schmied, Denise
Guymer, Robyn H.
Weber, Bernhard H. F.
Baird, Paul N.
SpringerLink (Online service)
- DOI
-
10.1186/s12974-016-0548-0
- URN
-
urn:nbn:de:1111-201604201664
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:51 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Grassmann, Felix
- Cantsilieris, Stuart
- Schulz-Kuhnt, Anja-Sabrina
- White, Stefan J.
- Richardson, Andrea J.
- Hewitt, Alex W.
- Vote, Brendan J.
- Schmied, Denise
- Guymer, Robyn H.
- Weber, Bernhard H. F.
- Baird, Paul N.
- SpringerLink (Online service)
Other Objects (12)
IFN-β immunomodulation and complement receptor analysis in a mouse model of age-related macular degeneration (AMD)
The complement system in age-related macular degeneration
NRF2-Associated Antioxidant Stress Response in Age-Related Macular Degeneration (AMD)
The role of complement in Age-related Macular Degeneration
Bevacizumab cured age-related macular degeneration (AMD) via down-regulate TLR2 pathway
Higher plasma levels of complement C3a, C4a and C5a increase the risk of subretinal fibrosis in neovascular age-related macular degeneration : Complement activation in AMD
The role of blood related inflammatory factors on age-related macular degeneration (AMD)
Age-Related Macular Degeneration (AMD) - Investigations into the Molecular Mechanisms of Genetic Associations
Mast cells in human choroid and their role in age-related macular degeneration (AMD)
Complement proteins and complement regulatory proteins are associated with age-related macular degeneration stage and treatment response
Epidemiology of age-related macular degeneration (AMD): associations with cardiovascular disease phenotypes and lipid factors
Age-related macular degeneration
IFN-β immunomodulation and complement receptor analysis in a mouse model of age-related macular degeneration (AMD)
The complement system in age-related macular degeneration
NRF2-Associated Antioxidant Stress Response in Age-Related Macular Degeneration (AMD)
The role of complement in Age-related Macular Degeneration
Bevacizumab cured age-related macular degeneration (AMD) via down-regulate TLR2 pathway
Higher plasma levels of complement C3a, C4a and C5a increase the risk of subretinal fibrosis in neovascular age-related macular degeneration : Complement activation in AMD
The role of blood related inflammatory factors on age-related macular degeneration (AMD)
Age-Related Macular Degeneration (AMD) - Investigations into the Molecular Mechanisms of Genetic Associations
Mast cells in human choroid and their role in age-related macular degeneration (AMD)
Complement proteins and complement regulatory proteins are associated with age-related macular degeneration stage and treatment response
Epidemiology of age-related macular degeneration (AMD): associations with cardiovascular disease phenotypes and lipid factors
Age-related macular degeneration
IFN-β immunomodulation and complement receptor analysis in a mouse model of age-related macular degeneration (AMD)
The complement system in age-related macular degeneration
NRF2-Associated Antioxidant Stress Response in Age-Related Macular Degeneration (AMD)
The role of complement in Age-related Macular Degeneration
Bevacizumab cured age-related macular degeneration (AMD) via down-regulate TLR2 pathway
Higher plasma levels of complement C3a, C4a and C5a increase the risk of subretinal fibrosis in neovascular age-related macular degeneration : Complement activation in AMD
The role of blood related inflammatory factors on age-related macular degeneration (AMD)
Age-Related Macular Degeneration (AMD) - Investigations into the Molecular Mechanisms of Genetic Associations
Mast cells in human choroid and their role in age-related macular degeneration (AMD)
Complement proteins and complement regulatory proteins are associated with age-related macular degeneration stage and treatment response
Epidemiology of age-related macular degeneration (AMD): associations with cardiovascular disease phenotypes and lipid factors