OncomiR-196 promotes an invasive phenotype in oral cancer through the NME4-JNK-TIMP1-MMP signaling pathway

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1476-4598

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: OncomiR-196 promotes an invasive phenotype in oral cancer through the NME4-JNK-TIMP1-MMP signaling pathway ; volume:13 ; number:1 ; day:19 ; month:9 ; year:2014 ; pages:1-14 ; date:12.2014
Molecular cancer ; 13, Heft 1 (19.9.2014), 1-14, 12.2014

Creator: Lu, Ya-Ching
Chang, Joseph T.
Liao, Chun-Ta
Kang, Chung-Jan
Huang, Shiang-Fu
Chen, I-How
Huang, Chi-Che
Huang, Yu-Chen
Chen, Wen-Ho
Tsai, Chi-Ying
Wang, Hung-Ming
Yen, Tzu-Chen
You, Guo-Rung
Chiang, Chang-Hsu
Cheng, Ann-Joy

Contributor: SpringerLink (Online service)

DOI: 10.1186/1476-4598-13-218

URN: urn:nbn:de:101:1-2019063020244257003338

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:02 AM CEST

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Associated

Lu, Ya-Ching
Chang, Joseph T.
Liao, Chun-Ta
Kang, Chung-Jan
Huang, Shiang-Fu
Chen, I-How
Huang, Chi-Che
Huang, Yu-Chen
Chen, Wen-Ho
Tsai, Chi-Ying
Wang, Hung-Ming
Yen, Tzu-Chen
You, Guo-Rung
Chiang, Chang-Hsu
Cheng, Ann-Joy
SpringerLink (Online service)

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OncomiR-196 promotes an invasive phenotype in oral cancer through the NME4-JNK-TIMP1-MMP signaling pathway

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Other Objects (12)

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NOX1 to NOX2 switch deactivates AMPK and induces invasive phenotype in colon cancer cells through overexpression of MMP-7

BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells

Triggering MSR1 promotes JNK‐mediated inflammation in IL‐4‐activated macrophages

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CAMSAP2 promotes colorectal cancer cell migration and invasion through activation of JNK/c-Jun/MMP-1 signaling pathway

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BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells

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hsa-mir-30c promotes the invasive phenotype of metastatic breast cancer cells by targeting NOV/CCN3

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Elevation of MMP-9 Levels Promotes Epileptogenesis After Traumatic Brain Injury

ANKRD49 promotes the metastasis of NSCLC via activating JNK-ATF2/c-Jun-MMP-2/9 axis

CAMSAP2 promotes colorectal cancer cell migration and invasion through activation of JNK/c-Jun/MMP-1 signaling pathway

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Targeting JNK pathway promotes human hematopoietic stem cell expansion

NOX1 to NOX2 switch deactivates AMPK and induces invasive phenotype in colon cancer cells through overexpression of MMP-7

BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells

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Elevation of MMP-9 Levels Promotes Epileptogenesis After Traumatic Brain Injury

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