BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2058-7716

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells ; volume:6 ; number:1 ; day:2 ; month:1 ; year:2020 ; pages:1-17 ; date:12.2020
Cell death discovery ; 6, Heft 1 (2.1.2020), 1-17, 12.2020

Creator: Janssen, Sanne Marlijn
Moscona, Roy
Elchebly, Mounib
Papadakis, Andreas Ioannis
Redpath, Margaret
Wang, Hangjun
Rubin, Eitan
van Kempen, Léon Cornelis
Spatz, Alan

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41420-019-0235-x

URN: urn:nbn:de:101:1-2021042119144939877402

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:44 AM CEST

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Associated

Janssen, Sanne Marlijn
Moscona, Roy
Elchebly, Mounib
Papadakis, Andreas Ioannis
Redpath, Margaret
Wang, Hangjun
Rubin, Eitan
van Kempen, Léon Cornelis
Spatz, Alan
SpringerLink (Online service)

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BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells

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Other Objects (12)

MCU controls melanoma progression through a redox‐controlled phenotype switch

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Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker

Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker

STAT3 – the switch of melanoma-associated NRAS mutations

Cigarette smoke and lipopolysaccharide induce a proliferative airway smooth muscle phenotype

ROR2, a driver of “phenotype switching” in melanoma?

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Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker

Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker

STAT3 – the switch of melanoma-associated NRAS mutations

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ROR2, a driver of “phenotype switching” in melanoma?

Plasmodium falciparum strains spontaneously switch invasion phenotype in suspension culture

DNA methylation subgroups in melanoma are associated with proliferative and immunological processes

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IgA nephropathy to proliferative glomerulonephritis with monoclonal IgAκ deposits: a pattern switch

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EGFR inhibitors switch keratinocytes from a proliferative to a differentiative phenotype affecting epidermal development and barrier function

Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker

Partial reprogramming of melanoma cells mimics the phenotype switch and reveals SNAI3 as a novel invasion-associated marker

STAT3 – the switch of melanoma-associated NRAS mutations

Cigarette smoke and lipopolysaccharide induce a proliferative airway smooth muscle phenotype

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