Understanding the role of epithelial MyD88 signals in the early development of Experimental Autoimmune Encephalomyelitis (EAE)
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
München, Ludwig-Maximilians-Universität, Dissertation, 2022
- Keyword
-
Tiermodell
Autoaggressionskrankheit
Maus
Immunsystem
Phänotyp
- Event
-
Veröffentlichung
- (where)
-
München
- (who)
-
Universitätsbibliothek der Ludwig-Maximilians-Universität
- (when)
-
2022
- Creator
- Contributor
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Kawakami, Naoto
- DOI
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10.5282/edoc.30754
- URN
-
urn:nbn:de:bvb:19-307547
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:54 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Solís González, Veronica E.
- Kawakami, Naoto
- Universitätsbibliothek der Ludwig-Maximilians-Universität
Time of origin
- 2022
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Functional characterization of the protein kinase MK2 in the pathogenesis of Experimental autoimmune encephalomyelitis (EAE)
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Assessment of mitochondrial dysfunction in experimental autoimmune encephalomyelitis (EAE) models of multiple sclerosis
Aspects of immunobiology of myelin oligodendrocyte glycoprotein (MOG) induced experimental autoimmune encephalomyelitis (EAE)
Microglial expression of Hdac1 and Hdac2 is dispensable for experimental autoimmune encephalomyelitis (EAE) progression
Molecular and histopathological validation of the combined Cup-EAE (cuprizone/experimental autoimmune encephalomyelitis) mouse model of multiple sclerosis
Experimentelle Autoimmune Encephalomyelitis - EAE - Läsionsentwicklung in subletal bestrahlten Tieren
The motorized RhoGAP myosin IXb (Myo9b) regulates experimental autoimmune encephalomyelitis (EAE)
Characterization of soluble adenylyl cyclease in experimental autoimmune encephalomyelitis (EAE)
Functional characterization of the protein kinase MK2 in the pathogenesis of Experimental autoimmune encephalomyelitis (EAE)
Evaluation of the fibroblast growth factor receptor 1 (FGFR1) in experimental autoimmune encephalomyelitis (EAE)
B-cell recruitment in spontaneous relapsing remitting (RR) experimental autoimmune encephalomyelitis (EAE)
The role of the transmembrane heparan sulfate proteoglycan, syndecan-1 (CD138), in murine experimental autoimmune encephalomyelitis (EAE)
Aspects of immunobiology of myelin oligodendrocyte glycoprotein (MOG) induced experimental autoimmune encephalomyelitis (EAE)
Assessment of mitochondrial dysfunction in experimental autoimmune encephalomyelitis (EAE) models of multiple sclerosis
Aspects of immunobiology of myelin oligodendrocyte glycoprotein (MOG) induced experimental autoimmune encephalomyelitis (EAE)
Microglial expression of Hdac1 and Hdac2 is dispensable for experimental autoimmune encephalomyelitis (EAE) progression
Molecular and histopathological validation of the combined Cup-EAE (cuprizone/experimental autoimmune encephalomyelitis) mouse model of multiple sclerosis
Experimentelle Autoimmune Encephalomyelitis - EAE - Läsionsentwicklung in subletal bestrahlten Tieren
The motorized RhoGAP myosin IXb (Myo9b) regulates experimental autoimmune encephalomyelitis (EAE)
Characterization of soluble adenylyl cyclease in experimental autoimmune encephalomyelitis (EAE)
Functional characterization of the protein kinase MK2 in the pathogenesis of Experimental autoimmune encephalomyelitis (EAE)
Evaluation of the fibroblast growth factor receptor 1 (FGFR1) in experimental autoimmune encephalomyelitis (EAE)
B-cell recruitment in spontaneous relapsing remitting (RR) experimental autoimmune encephalomyelitis (EAE)
The role of the transmembrane heparan sulfate proteoglycan, syndecan-1 (CD138), in murine experimental autoimmune encephalomyelitis (EAE)
Aspects of immunobiology of myelin oligodendrocyte glycoprotein (MOG) induced experimental autoimmune encephalomyelitis (EAE)
Assessment of mitochondrial dysfunction in experimental autoimmune encephalomyelitis (EAE) models of multiple sclerosis
Aspects of immunobiology of myelin oligodendrocyte glycoprotein (MOG) induced experimental autoimmune encephalomyelitis (EAE)
Microglial expression of Hdac1 and Hdac2 is dispensable for experimental autoimmune encephalomyelitis (EAE) progression