Multi-pathway DNA-repair reporters reveal competition between end-joining, single-strand annealing and homologous recombination at Cas9-induced DNA double-strand breaks
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-1723
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Multi-pathway DNA-repair reporters reveal competition between end-joining, single-strand annealing and homologous recombination at Cas9-induced DNA double-strand breaks ; volume:13 ; number:1 ; day:8 ; month:9 ; year:2022 ; pages:1-17 ; date:12.2022
Nature Communications ; 13, Heft 1 (8.9.2022), 1-17, 12.2022
- Creator
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van de Kooij, Bert
Kruswick, Alex
van Attikum, Haico
Yaffe, Michael B.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-022-32743-w
- URN
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urn:nbn:de:101:1-2022112521065482774717
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:25 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- van de Kooij, Bert
- Kruswick, Alex
- van Attikum, Haico
- Yaffe, Michael B.
- SpringerLink (Online service)
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One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1
Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair
High-complexity of DNA double-strand breaks is key for alternative end-joining choice
End resection: a key step in homologous recombination and DNA double-strand break repair
Radiation Induced DNA Double-Strand Breaks in Radiology
DNA double-strand break signaling and human disorders
APE1 promotes non-homologous end joining by initiating DNA double-strand break formation and decreasing ubiquitination of artemis following oxidative genotoxic stress
Presence of transient RNA-DNA hybrids at DNA double-strand break sites are necessary for efficient homologous-recombination mediated DNA repair
TOPOVIBL-REC114 interaction regulates meiotic DNA double-strand breaks
RNA involvement during non-homologous end joining of resected DNA double strand breaks in G1
One end to rule them all: Non-homologous end-joining and homologous recombination at DNA double-strand breaks
DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination
CtIP and MRN promote non-homologous end-joining of etoposide-induced DNA double-strand breaks in G1
Pyruvate kinase M2 regulates homologous recombination-mediated DNA double-strand break repair
High-complexity of DNA double-strand breaks is key for alternative end-joining choice
End resection: a key step in homologous recombination and DNA double-strand break repair
Radiation Induced DNA Double-Strand Breaks in Radiology
DNA double-strand break signaling and human disorders
APE1 promotes non-homologous end joining by initiating DNA double-strand break formation and decreasing ubiquitination of artemis following oxidative genotoxic stress
Presence of transient RNA-DNA hybrids at DNA double-strand break sites are necessary for efficient homologous-recombination mediated DNA repair
TOPOVIBL-REC114 interaction regulates meiotic DNA double-strand breaks