Aporphine and isoquinoline derivatives block glioblastoma cell stemness and enhance temozolomide cytotoxicity
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Aporphine and isoquinoline derivatives block glioblastoma cell stemness and enhance temozolomide cytotoxicity ; volume:12 ; number:1 ; day:7 ; month:12 ; year:2022 ; pages:1-17 ; date:12.2022
Scientific reports ; 12, Heft 1 (7.12.2022), 1-17, 12.2022
- Creator
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Rodrigues-Junior, Dorival Mendes
Raminelli, Cristiano
Hassanie, Haifa
Trossini, Gustavo Henrique Goulart
Perecim, Givago Prado
Caja, Laia
Moustakas, Aristidis
Vettore, André Luiz
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-022-25534-2
- URN
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urn:nbn:de:101:1-2023021721013098815047
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 11:03 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Rodrigues-Junior, Dorival Mendes
- Raminelli, Cristiano
- Hassanie, Haifa
- Trossini, Gustavo Henrique Goulart
- Perecim, Givago Prado
- Caja, Laia
- Moustakas, Aristidis
- Vettore, André Luiz
- SpringerLink (Online service)
Other Objects (12)
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Enhancing glioblastoma cytotoxicity through encapsulating O6-benzylguanine and temozolomide in PEGylated liposomal nanocarrier: an in vitro study
PPAR-γ agonists reactivate the ALDOC-NR2F1 axis to enhance sensitivity to temozolomide and suppress glioblastoma progression
Temozolomide promotes genomic and phenotypic changes in glioblastoma cells
p53/E2F7 axis promotes temozolomide chemoresistance in glioblastoma multiforme
Considering the experimental use of temozolomide in glioblastoma research
Toxicity after radiochemotherapy for glioblastoma using temozolomide - a retrospective evaluation
Combined effects of niclosamide and temozolomide against human glioblastoma tumorspheres
One-carbon-mediated purine synthesis underlies temozolomide resistance in glioblastoma
Inhibition of gap junctions sensitizes primary glioblastoma cells for temozolomide
Glioblastoma stem cells deliver ABCB4 transcribed by ATF3 via exosomes conferring glioblastoma resistance to temozolomide
Accelerated hyperfractionation plus temozolomide in glioblastoma
Etomoxir, a carnitine palmitoyltransferase 1 inhibitor, combined with temozolomide reduces stemness and invasiveness in patient-derived glioblastoma tumorspheres
Enhancing glioblastoma cytotoxicity through encapsulating O6-benzylguanine and temozolomide in PEGylated liposomal nanocarrier: an in vitro study
PPAR-γ agonists reactivate the ALDOC-NR2F1 axis to enhance sensitivity to temozolomide and suppress glioblastoma progression
Temozolomide promotes genomic and phenotypic changes in glioblastoma cells
p53/E2F7 axis promotes temozolomide chemoresistance in glioblastoma multiforme
Considering the experimental use of temozolomide in glioblastoma research
Toxicity after radiochemotherapy for glioblastoma using temozolomide - a retrospective evaluation
Combined effects of niclosamide and temozolomide against human glioblastoma tumorspheres
One-carbon-mediated purine synthesis underlies temozolomide resistance in glioblastoma
Inhibition of gap junctions sensitizes primary glioblastoma cells for temozolomide
Glioblastoma stem cells deliver ABCB4 transcribed by ATF3 via exosomes conferring glioblastoma resistance to temozolomide
Accelerated hyperfractionation plus temozolomide in glioblastoma
Etomoxir, a carnitine palmitoyltransferase 1 inhibitor, combined with temozolomide reduces stemness and invasiveness in patient-derived glioblastoma tumorspheres
Enhancing glioblastoma cytotoxicity through encapsulating O6-benzylguanine and temozolomide in PEGylated liposomal nanocarrier: an in vitro study
PPAR-γ agonists reactivate the ALDOC-NR2F1 axis to enhance sensitivity to temozolomide and suppress glioblastoma progression
Temozolomide promotes genomic and phenotypic changes in glioblastoma cells
p53/E2F7 axis promotes temozolomide chemoresistance in glioblastoma multiforme
Considering the experimental use of temozolomide in glioblastoma research
Toxicity after radiochemotherapy for glioblastoma using temozolomide - a retrospective evaluation
Combined effects of niclosamide and temozolomide against human glioblastoma tumorspheres
One-carbon-mediated purine synthesis underlies temozolomide resistance in glioblastoma
Inhibition of gap junctions sensitizes primary glioblastoma cells for temozolomide