Cre recombinase promotes leukemogenesis in the presence of both homozygous and heterozygous FLT3-ITD
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Cre recombinase promotes leukemogenesis in the presence of both homozygous and heterozygous FLT3-ITD ; volume:38 ; number:6 ; day:8 ; month:5 ; year:2024 ; pages:1437-1439 ; date:6.2024
Leukemia ; 38, Heft 6 (8.5.2024), 1437-1439, 6.2024
- Creator
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Yang, Min
Ma, Zhiyuan
Wang, Chonggang
Agca, Muammer Cihan
Liu, Hongyun
Huang, Kezhi
Glage, Silke
Rumpel, Regina
Gerbaulet, Alexander
Roers, Axel
Liu, Xuemei
Noyan, Fatih
Neuhoff, Nils von
Ganser, Arnold
Liu, Ligen
Yun, Haiyang
Li, Zhixiong
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41375-024-02259-x
- URN
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urn:nbn:de:101:1-2408102103227.718497426437
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:59 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Yang, Min
- Ma, Zhiyuan
- Wang, Chonggang
- Agca, Muammer Cihan
- Liu, Hongyun
- Huang, Kezhi
- Glage, Silke
- Rumpel, Regina
- Gerbaulet, Alexander
- Roers, Axel
- Liu, Xuemei
- Noyan, Fatih
- Neuhoff, Nils von
- Ganser, Arnold
- Liu, Ligen
- Yun, Haiyang
- Li, Zhixiong
- SpringerLink (Online service)
Other Objects (12)
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FLT3-ITD regulated miRs in AML
RSK1 dependency in FLT3-ITD acute myeloid leukemia
Role of FLT3 ITD PTPRC signaling in bone remodeling
CCL5 mediates target-kinase independent resistance to FLT3 inhibitors in FLT3-ITD-positive AML
Distinct Metabolic features differentiating FLT3-ITD AML from FLT3-WT childhood Acute Myeloid Leukemia
Identification of Protein Tyrosine Phosphatases regulating FLT3 activity and of PTPs regulated by FLT3 ITD
Autophagy activation mediates resistance to FLT3 inhibitors in acute myeloid leukemia with FLT3-ITD mutation
Dendritic cell dysfunctions in AML patients with FLT3 ITD mutations
Effekte von FLT3-ITD Inhibitoren auf Chemotherapie resistente AML Zelllinien
The role of STAT5 in FLT3-ITD-mediated leukemogenesis
Role of SOCS proteins in FLT3-ITD and BCR-ABL mediated leukemogenesis
Abnormal Localization and Accumulation of FLT3-ITD, a Mutant Receptor Tyrosine Kinase Involved in Leukemogenesis
FLT3-ITD regulated miRs in AML
RSK1 dependency in FLT3-ITD acute myeloid leukemia
Role of FLT3 ITD PTPRC signaling in bone remodeling
CCL5 mediates target-kinase independent resistance to FLT3 inhibitors in FLT3-ITD-positive AML
Distinct Metabolic features differentiating FLT3-ITD AML from FLT3-WT childhood Acute Myeloid Leukemia
Identification of Protein Tyrosine Phosphatases regulating FLT3 activity and of PTPs regulated by FLT3 ITD
Autophagy activation mediates resistance to FLT3 inhibitors in acute myeloid leukemia with FLT3-ITD mutation
Dendritic cell dysfunctions in AML patients with FLT3 ITD mutations
Effekte von FLT3-ITD Inhibitoren auf Chemotherapie resistente AML Zelllinien
The role of STAT5 in FLT3-ITD-mediated leukemogenesis
Role of SOCS proteins in FLT3-ITD and BCR-ABL mediated leukemogenesis
Abnormal Localization and Accumulation of FLT3-ITD, a Mutant Receptor Tyrosine Kinase Involved in Leukemogenesis
FLT3-ITD regulated miRs in AML
RSK1 dependency in FLT3-ITD acute myeloid leukemia
Role of FLT3 ITD PTPRC signaling in bone remodeling
CCL5 mediates target-kinase independent resistance to FLT3 inhibitors in FLT3-ITD-positive AML
Distinct Metabolic features differentiating FLT3-ITD AML from FLT3-WT childhood Acute Myeloid Leukemia
Identification of Protein Tyrosine Phosphatases regulating FLT3 activity and of PTPs regulated by FLT3 ITD
Autophagy activation mediates resistance to FLT3 inhibitors in acute myeloid leukemia with FLT3-ITD mutation
Dendritic cell dysfunctions in AML patients with FLT3 ITD mutations