Abnormal Localization and Accumulation of FLT3-ITD, a Mutant Receptor Tyrosine Kinase Involved in Leukemogenesis
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Erschienen in
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In: Cells Tissues Organs 2008;188:225–235, ISSN: 1422-6405
- Ereignis
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Veröffentlichung
- (wo)
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Dresden
- (wer)
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Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden
- (wann)
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2014
- Urheber
- URN
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urn:nbn:de:bsz:14-qucosa-136167
- Rechteinformation
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
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14.08.2025, 10:48 MESZ
Datenpartner
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Beteiligte
- Koch, Sina
- Jacobi, Angela
- Ryser, Martin
- Ehninger, Gerhard
- Thiede, Christian
- Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden
Entstanden
- 2014
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Cre recombinase promotes leukemogenesis in the presence of both homozygous and heterozygous FLT3-ITD
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Identification of Protein Tyrosine Phosphatases regulating FLT3 activity and of PTPs regulated by FLT3 ITD
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Synergistic effect of FMS-like tyrosine kinase-3 (FLT3) inhibitors combined with a CDK7 inhibitor in FLT3-ITD-mutated acute myeloid leukemia
Tyrosine 842 in the activation loop is required for full transformation by the oncogenic mutant FLT3-ITD
Role of FLT3 ITD PTPRC signaling in bone remodeling
The role of STAT5 in FLT3-ITD-mediated leukemogenesis
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Cre recombinase promotes leukemogenesis in the presence of both homozygous and heterozygous FLT3-ITD
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Synergistic effect of FMS-like tyrosine kinase-3 (FLT3) inhibitors combined with a CDK7 inhibitor in FLT3-ITD-mutated acute myeloid leukemia
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