A temporally dynamic Foxp3 autoregulatory transcriptional circuit controls the effector Treg programme
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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A temporally dynamic Foxp3 autoregulatory transcriptional circuit controls the effector Treg programme ; volume:37 ; number:16 ; year:2018 ; extent:16
The EMBO journal / European Molecular Biology Organization ; 37, Heft 16 (2018) (gesamt 16)
- Creator
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Bending, David
Paduraru, Alina
Ducker, Catherine B.
Prieto Martín, Paz
Crompton, Tessa
Ono, Masahiro
- DOI
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10.15252/embj.201899013
- URN
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urn:nbn:de:101:1-2022083108284371975089
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:34 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Bending, David
- Paduraru, Alina
- Ducker, Catherine B.
- Prieto Martín, Paz
- Crompton, Tessa
- Ono, Masahiro
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CD4+CCR8+ Tregs in ovarian cancer: a potential effector Tregs for immune regulation
Foxp3 + Treg-derived IL-10 promotes colorectal cancer-derived lung metastasis
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
GARP derived TGFβ promotes acetylation-mediated Foxp3 protein stabilization and Treg functionality
The deubiquitinase USP44 promotes Treg function during inflammation by preventing FOXP3 degradation
Identifizierung von Treg-spezifischen Signaltransduktionswegen und die Rolle der subzellulären Lokalisation von FOXP3
The functional adaptation of effector Foxp3 + regulatory T cells to pulmonary inflammation
CD44 expression positively correlates with Foxp3 expression and suppressive function of CD4+ Treg cells
IL-23 stabilizes an effector Treg cell program in the tumor microenvironment
The function of RORγt+Foxp3+ biTregs in glomerulonephritis : Die Rolle von RORγt+Foxp3+ biTregs im Rahmen der Glomerulonephritis
Maintenance and stability of Foxp3+ regulatory T (Treg) cells
CD4+CCR8+ Tregs in ovarian cancer: a potential effector Tregs for immune regulation
Foxp3 + Treg-derived IL-10 promotes colorectal cancer-derived lung metastasis
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
GARP derived TGFβ promotes acetylation-mediated Foxp3 protein stabilization and Treg functionality
The deubiquitinase USP44 promotes Treg function during inflammation by preventing FOXP3 degradation
Identifizierung von Treg-spezifischen Signaltransduktionswegen und die Rolle der subzellulären Lokalisation von FOXP3
The functional adaptation of effector Foxp3 + regulatory T cells to pulmonary inflammation
CD44 expression positively correlates with Foxp3 expression and suppressive function of CD4+ Treg cells
IL-23 stabilizes an effector Treg cell program in the tumor microenvironment
The function of RORγt+Foxp3+ biTregs in glomerulonephritis : Die Rolle von RORγt+Foxp3+ biTregs im Rahmen der Glomerulonephritis
Maintenance and stability of Foxp3+ regulatory T (Treg) cells
CD4+CCR8+ Tregs in ovarian cancer: a potential effector Tregs for immune regulation
Foxp3 + Treg-derived IL-10 promotes colorectal cancer-derived lung metastasis
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
Characterization of inflammatory RORγt+ (Th17) and regulatory Foxp3+ (Treg) T cell populations
GARP derived TGFβ promotes acetylation-mediated Foxp3 protein stabilization and Treg functionality
The deubiquitinase USP44 promotes Treg function during inflammation by preventing FOXP3 degradation
Identifizierung von Treg-spezifischen Signaltransduktionswegen und die Rolle der subzellulären Lokalisation von FOXP3
The functional adaptation of effector Foxp3 + regulatory T cells to pulmonary inflammation
CD44 expression positively correlates with Foxp3 expression and suppressive function of CD4+ Treg cells