Compensatory T-type Ca2+ channel activity alters D2-autoreceptor responses of Substantia nigra dopamine neurons from Cav1.3 L-type Ca2+ channel KO mice
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
-
Englisch
- Bibliographic citation
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In: Scientific Reports ; 5 (2015). - Art.-Nr. 13688. - eISSN 2045-2322
- Keyword
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Calciumkanal
Substantia nigra
- Event
-
Veröffentlichung
- (where)
-
Ulm
- (who)
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Universität Ulm
- (when)
-
2015
- Creator
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Pötschke, Christina
Dragicevic, Elena
Duda, Johanna
Benkert, Julia
Dougalis, Antonios
Dezio, Roberta
Snutch, Terrance P.
Striessnig, Jörg
Liss, Birgit
- DOI
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10.18725/OPARU-49117
- URN
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urn:nbn:de:bsz:289-oparu-49193-2
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:48 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Pötschke, Christina
- Dragicevic, Elena
- Duda, Johanna
- Benkert, Julia
- Dougalis, Antonios
- Dezio, Roberta
- Snutch, Terrance P.
- Striessnig, Jörg
- Liss, Birgit
- Universität Ulm
Time of origin
- 2015
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CaV1.3 L-type Ca2+ channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na+ current
Blockade of L-type Ca2+ channel attenuates doxorubicin-induced cardiomyopathy via suppression of CaMKII-NF-κB pathway
Optical control of L-type Ca2+ channels using a diltiazem photoswitch
Structural determinants of CaV1.3 L-type calcium channel gating
Coupling of SK channels, L-type Ca2+ channels, and ryanodine receptors in cardiomyocytes
C-terminal splicing reveals intramolecular gating modulation in CaV1.3 L-type Ca2+ channels
Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity
L type Ca2+ channel blockers prevent oxaliplatin-induced cold hyperalgesia and TRPM8 overexpression in rats
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Impaired L-type Ca2+ channel function in the dystrophic heart
Functional analysis of L-type voltagegated Ca2+ channel isoforms Cav1.2 and Cav1.3 in NG2 glia
In vivo and in vitro analysis of the retinal voltage dependent L-type Ca2+channel Ca 1.4 function
CaV1.3 L-type Ca2+ channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na+ current
Blockade of L-type Ca2+ channel attenuates doxorubicin-induced cardiomyopathy via suppression of CaMKII-NF-κB pathway
Optical control of L-type Ca2+ channels using a diltiazem photoswitch
Structural determinants of CaV1.3 L-type calcium channel gating
Coupling of SK channels, L-type Ca2+ channels, and ryanodine receptors in cardiomyocytes
C-terminal splicing reveals intramolecular gating modulation in CaV1.3 L-type Ca2+ channels
Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity
L type Ca2+ channel blockers prevent oxaliplatin-induced cold hyperalgesia and TRPM8 overexpression in rats
L-type voltage-dependent Ca2+ channels expression involved in pre-neoplastic transformation of breast cancer
Impaired L-type Ca2+ channel function in the dystrophic heart
Functional analysis of L-type voltagegated Ca2+ channel isoforms Cav1.2 and Cav1.3 in NG2 glia
In vivo and in vitro analysis of the retinal voltage dependent L-type Ca2+channel Ca 1.4 function
CaV1.3 L-type Ca2+ channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na+ current
Blockade of L-type Ca2+ channel attenuates doxorubicin-induced cardiomyopathy via suppression of CaMKII-NF-κB pathway
Optical control of L-type Ca2+ channels using a diltiazem photoswitch
Structural determinants of CaV1.3 L-type calcium channel gating
Coupling of SK channels, L-type Ca2+ channels, and ryanodine receptors in cardiomyocytes
C-terminal splicing reveals intramolecular gating modulation in CaV1.3 L-type Ca2+ channels
Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity
L type Ca2+ channel blockers prevent oxaliplatin-induced cold hyperalgesia and TRPM8 overexpression in rats