CaV1.3 L-type Ca2+ channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na+ current
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
CaV1.3 L-type Ca2+ channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na+ current ; volume:7 ; number:1 ; day:11 ; month:8 ; year:2017 ; pages:1-12 ; date:12.2017
Scientific reports ; 7, Heft 1 (11.8.2017), 1-12, 12.2017
- Classification
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Biowissenschaften, Biologie
- Creator
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Toyoda, Futoshi
Mesirca, Pietro
Dubel, Stefan
Ding, Wei-Guang
Striessnig, Joerg
Mangoni, Matteo E.
Matsuura, Hiroshi
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-017-08191-8
- URN
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urn:nbn:de:101:1-2019070722094641796297
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:33 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Toyoda, Futoshi
- Mesirca, Pietro
- Dubel, Stefan
- Ding, Wei-Guang
- Striessnig, Joerg
- Mangoni, Matteo E.
- Matsuura, Hiroshi
- SpringerLink (Online service)
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C-terminal splicing reveals intramolecular gating modulation in CaV1.3 L-type Ca2+ channels
Generating dendritic Ca2+ spikes with different models of Ca2+ buffering in cerebellar Purkinje cells
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Compensatory T-type Ca2+ channel activity alters D2-autoreceptor responses of Substantia nigra dopamine neurons from Cav1.3 L-type Ca2+ channel KO mice
Local hyperactivation of L-type Ca2+ channels increases spontaneous Ca2+ release activity and cellular hypertrophy in right ventricular myocytes from heart failure rats
In vivo and in vitro analysis of the retinal voltage dependent L-type Ca2+channel Ca 1.4 function
CXCL12-induced neurotoxicity critically depends on NMDA receptor-gated and l-type Ca2+ channels upstream of p38 MAPK
Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity
Impaired L-type Ca2+ channel function in the dystrophic heart
Optical control of L-type Ca2+ channels using a diltiazem photoswitch
Coupling of SK channels, L-type Ca2+ channels, and ryanodine receptors in cardiomyocytes
C-terminal splicing reveals intramolecular gating modulation in CaV1.3 L-type Ca2+ channels
Generating dendritic Ca2+ spikes with different models of Ca2+ buffering in cerebellar Purkinje cells
L-type voltage-dependent Ca2+ channels expression involved in pre-neoplastic transformation of breast cancer
Functional analysis of L-type voltagegated Ca2+ channel isoforms Cav1.2 and Cav1.3 in NG2 glia
Compensatory T-type Ca2+ channel activity alters D2-autoreceptor responses of Substantia nigra dopamine neurons from Cav1.3 L-type Ca2+ channel KO mice
Local hyperactivation of L-type Ca2+ channels increases spontaneous Ca2+ release activity and cellular hypertrophy in right ventricular myocytes from heart failure rats
In vivo and in vitro analysis of the retinal voltage dependent L-type Ca2+channel Ca 1.4 function
CXCL12-induced neurotoxicity critically depends on NMDA receptor-gated and l-type Ca2+ channels upstream of p38 MAPK
Concomitant genetic ablation of L-type Cav1.3 (α1D) and T-type Cav3.1 (α1G) Ca2+ channels disrupts heart automaticity
Impaired L-type Ca2+ channel function in the dystrophic heart
Optical control of L-type Ca2+ channels using a diltiazem photoswitch
Coupling of SK channels, L-type Ca2+ channels, and ryanodine receptors in cardiomyocytes
C-terminal splicing reveals intramolecular gating modulation in CaV1.3 L-type Ca2+ channels
Generating dendritic Ca2+ spikes with different models of Ca2+ buffering in cerebellar Purkinje cells
L-type voltage-dependent Ca2+ channels expression involved in pre-neoplastic transformation of breast cancer
Functional analysis of L-type voltagegated Ca2+ channel isoforms Cav1.2 and Cav1.3 in NG2 glia
Compensatory T-type Ca2+ channel activity alters D2-autoreceptor responses of Substantia nigra dopamine neurons from Cav1.3 L-type Ca2+ channel KO mice
Local hyperactivation of L-type Ca2+ channels increases spontaneous Ca2+ release activity and cellular hypertrophy in right ventricular myocytes from heart failure rats
In vivo and in vitro analysis of the retinal voltage dependent L-type Ca2+channel Ca 1.4 function
CXCL12-induced neurotoxicity critically depends on NMDA receptor-gated and l-type Ca2+ channels upstream of p38 MAPK