SRSF10 stabilizes CDC25A by triggering exon 6 skipping to promote hepatocarcinogenesis

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1756-9966

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: SRSF10 stabilizes CDC25A by triggering exon 6 skipping to promote hepatocarcinogenesis ; volume:41 ; number:1 ; day:20 ; month:12 ; year:2022 ; pages:1-19 ; date:12.2022
Journal of experimental & clinical cancer research ; 41, Heft 1 (20.12.2022), 1-19, 12.2022

Creator: Liu, Xiaoming
Zheng, Yongqiang
Xiao, Mengqing
Chen, Xingyu
Zhu, Yuxing
Xu, Canxia
Wang, Fen
Liu, Zexian
Cao, Ke

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13046-022-02558-0

URN: urn:nbn:de:101:1-2023030221215173218527

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:51 AM CEST

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Associated

Liu, Xiaoming
Zheng, Yongqiang
Xiao, Mengqing
Chen, Xingyu
Zhu, Yuxing
Xu, Canxia
Wang, Fen
Liu, Zexian
Cao, Ke
SpringerLink (Online service)

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SRSF10 stabilizes CDC25A by triggering exon 6 skipping to promote hepatocarcinogenesis

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Other Objects (12)

Exon skipping for DMD

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Exon skipping as a therapeutic strategy in dysferlinopathy

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Unexpected consequences: exon skipping caused by CRISPR-generated mutations

CRISPR/Cas9-mediated genome editing induces exon skipping by alternative splicing or exon deletion

A compatible exon-exon junction database for the identification of exon skipping events using tandem mass spectrum data

Antisense-induced exon skipping for duplications in Duchenne muscular dystrophy

Exon skipping-rich transcriptomes of animals reflect the significance of exon-shuffling in metazoan proteome evolution

CRISPR-induced exon skipping is dependent on premature termination codon mutations

Dual exon skipping in myostatin and dystrophin for Duchenne muscular dystrophy

Reverse complementary matches simultaneously promote both back-splicing and exon-skipping

Exon skipping for DMD

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Exon skipping as a therapeutic strategy in dysferlinopathy

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Unexpected consequences: exon skipping caused by CRISPR-generated mutations

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Dual exon skipping in myostatin and dystrophin for Duchenne muscular dystrophy

Reverse complementary matches simultaneously promote both back-splicing and exon-skipping

Exon skipping for DMD

Elevated nuclear TDP-43 induces constitutive exon skipping

Exon skipping as a therapeutic strategy in dysferlinopathy

Applying Lessons Learned from Developing Exon Skipping for Duchenne to Developing Individualized Exon Skipping Therapy for Patients with Neurodegenerative Diseases

Unexpected consequences: exon skipping caused by CRISPR-generated mutations

CRISPR/Cas9-mediated genome editing induces exon skipping by alternative splicing or exon deletion

A compatible exon-exon junction database for the identification of exon skipping events using tandem mass spectrum data

Antisense-induced exon skipping for duplications in Duchenne muscular dystrophy

Exon skipping-rich transcriptomes of animals reflect the significance of exon-shuffling in metazoan proteome evolution

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Dual exon skipping in myostatin and dystrophin for Duchenne muscular dystrophy

Reverse complementary matches simultaneously promote both back-splicing and exon-skipping

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