CRISPR-induced exon skipping is dependent on premature termination codon mutations
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1474-760X
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
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CRISPR-induced exon skipping is dependent on premature termination codon mutations ; volume:19 ; number:1 ; day:17 ; month:10 ; year:2018 ; pages:1-5 ; date:12.2018
Genome biology ; 19, Heft 1 (17.10.2018), 1-5, 12.2018
- Urheber
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Sui, Tingting
- Beteiligte Personen und Organisationen
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Song, Yuning
Liu, Zhiquan
Chen, Mao
Deng, Jichao
Xu, Yuanyuan
Lai, Liangxue
Li, Zhanjun
SpringerLink (Online service)
- DOI
-
10.1186/s13059-018-1532-z
- URN
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urn:nbn:de:101:1-2018112515193583666163
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:59 MESZ
Datenpartner
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Beteiligte
- Sui, Tingting
- Song, Yuning
- Liu, Zhiquan
- Chen, Mao
- Deng, Jichao
- Xu, Yuanyuan
- Lai, Liangxue
- Li, Zhanjun
- SpringerLink (Online service)
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Variable expression of cerebral cavernous malformations in carriers of a premature termination codon in exon 17 of the Krit1 gene
Effect of small molecule eRF3 degraders on premature termination codon readthrough
Ataluren suppresses a premature termination codon in an MPS I-H mouse
Efficiency of translation termination in humans is highly dependent upon nucleotides in the neighbourhood of a (premature) termination codon
Elevated nuclear TDP-43 induces constitutive exon skipping
Exon skipping as a therapeutic strategy in dysferlinopathy
Applying Lessons Learned from Developing Exon Skipping for Duchenne to Developing Individualized Exon Skipping Therapy for Patients with Neurodegenerative Diseases
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Premature termination codon readthrough upregulates progranulin expression and improves lysosomal function in preclinical models of GRN deficiency
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Variable expression of cerebral cavernous malformations in carriers of a premature termination codon in exon 17 of the Krit1 gene
Effect of small molecule eRF3 degraders on premature termination codon readthrough
Ataluren suppresses a premature termination codon in an MPS I-H mouse
Efficiency of translation termination in humans is highly dependent upon nucleotides in the neighbourhood of a (premature) termination codon
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Premature termination codon readthrough upregulates progranulin expression and improves lysosomal function in preclinical models of GRN deficiency