CRISPR-induced exon skipping is dependent on premature termination codon mutations
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1474-760X
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
CRISPR-induced exon skipping is dependent on premature termination codon mutations ; volume:19 ; number:1 ; day:17 ; month:10 ; year:2018 ; pages:1-5 ; date:12.2018
Genome biology ; 19, Heft 1 (17.10.2018), 1-5, 12.2018
- Creator
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Sui, Tingting
- Contributor
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Song, Yuning
Liu, Zhiquan
Chen, Mao
Deng, Jichao
Xu, Yuanyuan
Lai, Liangxue
Li, Zhanjun
SpringerLink (Online service)
- DOI
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10.1186/s13059-018-1532-z
- URN
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urn:nbn:de:101:1-2018112515193583666163
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:59 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Sui, Tingting
- Song, Yuning
- Liu, Zhiquan
- Chen, Mao
- Deng, Jichao
- Xu, Yuanyuan
- Lai, Liangxue
- Li, Zhanjun
- SpringerLink (Online service)
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Effect of small molecule eRF3 degraders on premature termination codon readthrough
Ataluren suppresses a premature termination codon in an MPS I-H mouse
Efficiency of translation termination in humans is highly dependent upon nucleotides in the neighbourhood of a (premature) termination codon
Elevated nuclear TDP-43 induces constitutive exon skipping
Exon skipping as a therapeutic strategy in dysferlinopathy
Applying Lessons Learned from Developing Exon Skipping for Duchenne to Developing Individualized Exon Skipping Therapy for Patients with Neurodegenerative Diseases
Unexpected consequences: exon skipping caused by CRISPR-generated mutations
Premature termination codon readthrough upregulates progranulin expression and improves lysosomal function in preclinical models of GRN deficiency
CRISPR/Cas9-mediated genome editing induces exon skipping by alternative splicing or exon deletion
The role of nucleotide composition in premature termination codon recognition
Exon skipping for DMD
Variable expression of cerebral cavernous malformations in carriers of a premature termination codon in exon 17 of the Krit1 gene
Effect of small molecule eRF3 degraders on premature termination codon readthrough
Ataluren suppresses a premature termination codon in an MPS I-H mouse
Efficiency of translation termination in humans is highly dependent upon nucleotides in the neighbourhood of a (premature) termination codon
Elevated nuclear TDP-43 induces constitutive exon skipping
Exon skipping as a therapeutic strategy in dysferlinopathy
Applying Lessons Learned from Developing Exon Skipping for Duchenne to Developing Individualized Exon Skipping Therapy for Patients with Neurodegenerative Diseases
Unexpected consequences: exon skipping caused by CRISPR-generated mutations
Premature termination codon readthrough upregulates progranulin expression and improves lysosomal function in preclinical models of GRN deficiency
CRISPR/Cas9-mediated genome editing induces exon skipping by alternative splicing or exon deletion
The role of nucleotide composition in premature termination codon recognition
Exon skipping for DMD
Variable expression of cerebral cavernous malformations in carriers of a premature termination codon in exon 17 of the Krit1 gene
Effect of small molecule eRF3 degraders on premature termination codon readthrough
Ataluren suppresses a premature termination codon in an MPS I-H mouse
Efficiency of translation termination in humans is highly dependent upon nucleotides in the neighbourhood of a (premature) termination codon
Elevated nuclear TDP-43 induces constitutive exon skipping
Exon skipping as a therapeutic strategy in dysferlinopathy
Applying Lessons Learned from Developing Exon Skipping for Duchenne to Developing Individualized Exon Skipping Therapy for Patients with Neurodegenerative Diseases
Unexpected consequences: exon skipping caused by CRISPR-generated mutations
Premature termination codon readthrough upregulates progranulin expression and improves lysosomal function in preclinical models of GRN deficiency