In vivo analysis of calcium-initiated axon degeneration in an animal model of MS
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
München, Ludwig-Maximilians-Universität, Dissertation, 2018
- Keyword
-
Degeneration
Calcium
Calpain
Multiple sclerosis
- Event
-
Veröffentlichung
- (where)
-
München
- (who)
-
Universitätsbibliothek der Ludwig-Maximilians-Universität
- (when)
-
2018
- Creator
- Contributor
-
Kerschensteiner, Martin
- URN
-
urn:nbn:de:bvb:19-230357
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:53 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wesolowski, Marta
- Kerschensteiner, Martin
- Universitätsbibliothek der Ludwig-Maximilians-Universität
Time of origin
- 2018
Other Objects (12)
Mitochondrial damage and axon degeneration in chronic neuroinflammation
Involvement of N-type voltage dependent calcium channels in axon degeneration during experimental autoimmune optic neuritis
Wallerian degeneration and Wallerian related axon loss in disease
Wallerian degeneration and Wallerian-related axon loss in disease
The role of calcium in axonal degeneration in an animal model of multiple sclerosis
Dysregulated IGFBP5 expression causes axon degeneration and motoneuron loss in diabetic neuropathy
Analysis of Axon Outgrowth and Calcium Responses in Locust Neurons
Norrin protects optic nerve axons from degeneration in a mouse model of glaucoma
Fate of Oligodendrocytes during Retinal Axon Degeneration and Regeneration in the Goldfish Visual Pathway
Local axonal function of STAT3 rescues axon degeneration in the pmn model of motoneuron disease
Early Degeneration of Both Dopaminergic and Serotonergic Axons – A Common Mechanism in Parkinson’s Disease
Die Effekte des Wlds-Gens und der Gabe von Nikotinamid auf akute und chronisch-sekundäre Axon-Degeneration
Mitochondrial damage and axon degeneration in chronic neuroinflammation
Involvement of N-type voltage dependent calcium channels in axon degeneration during experimental autoimmune optic neuritis
Wallerian degeneration and Wallerian related axon loss in disease
Wallerian degeneration and Wallerian-related axon loss in disease
The role of calcium in axonal degeneration in an animal model of multiple sclerosis
Dysregulated IGFBP5 expression causes axon degeneration and motoneuron loss in diabetic neuropathy
Analysis of Axon Outgrowth and Calcium Responses in Locust Neurons
Norrin protects optic nerve axons from degeneration in a mouse model of glaucoma
Fate of Oligodendrocytes during Retinal Axon Degeneration and Regeneration in the Goldfish Visual Pathway
Local axonal function of STAT3 rescues axon degeneration in the pmn model of motoneuron disease
Early Degeneration of Both Dopaminergic and Serotonergic Axons – A Common Mechanism in Parkinson’s Disease
Die Effekte des Wlds-Gens und der Gabe von Nikotinamid auf akute und chronisch-sekundäre Axon-Degeneration
Mitochondrial damage and axon degeneration in chronic neuroinflammation
Involvement of N-type voltage dependent calcium channels in axon degeneration during experimental autoimmune optic neuritis
Wallerian degeneration and Wallerian related axon loss in disease
Wallerian degeneration and Wallerian-related axon loss in disease
The role of calcium in axonal degeneration in an animal model of multiple sclerosis
Dysregulated IGFBP5 expression causes axon degeneration and motoneuron loss in diabetic neuropathy
Analysis of Axon Outgrowth and Calcium Responses in Locust Neurons
Norrin protects optic nerve axons from degeneration in a mouse model of glaucoma
Fate of Oligodendrocytes during Retinal Axon Degeneration and Regeneration in the Goldfish Visual Pathway
Local axonal function of STAT3 rescues axon degeneration in the pmn model of motoneuron disease
Early Degeneration of Both Dopaminergic and Serotonergic Axons – A Common Mechanism in Parkinson’s Disease