Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1742-2094
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice ; volume:11 ; number:1 ; day:3 ; month:3 ; year:2014 ; pages:1-14 ; date:12.2014
Journal of neuroinflammation ; 11, Heft 1 (3.3.2014), 1-14, 12.2014
- Creator
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Niesman, Ingrid R.
- Contributor
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Schilling, Jan M.
Shapiro, Lee A.
Kellerhals, Sarah E.
Bonds, Jacqueline A.
Kleschevnikov, Alexander M.
Cui, Weihua
Voong, April
Krajewski, Stan
Ali, Sameh S.
Roth, David M.
Patel, Hemal H.
Patel, Piyush M.
Head, Brian P.
SpringerLink (Online service)
- DOI
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10.1186/1742-2094-11-39
- URN
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urn:nbn:de:1111-2016032337480
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:44 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Niesman, Ingrid R.
- Schilling, Jan M.
- Shapiro, Lee A.
- Kellerhals, Sarah E.
- Bonds, Jacqueline A.
- Kleschevnikov, Alexander M.
- Cui, Weihua
- Voong, April
- Krajewski, Stan
- Ali, Sameh S.
- Roth, David M.
- Patel, Hemal H.
- Patel, Piyush M.
- Head, Brian P.
- SpringerLink (Online service)
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Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen
Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen
Caveolin-1 enhances resveratrol-mediated cytotoxicity and transport in a hepatocellular carcinoma model
Caveolin-2 associates with intracellular chlamydial inclusions independently of caveolin-1
Hyperglycemia enhances brain susceptibility to lipopolysaccharide-induced neuroinflammation via astrocyte reprogramming
Klonierung des muskelspezifischen Caveolin-3 und transkriptionelle Regulation des Caveolin-3-Promoters
Spatial transcriptomics and neurofilament light chain reveal changes in lesion patterns in murine autoimmune neuroinflammation
Complement C3 Enhances LPS-Elicited Neuroinflammation and Neurodegeneration Via the Mac1/NOX2 Pathway
NLRP3 ablation enhances tolerance in heat stroke pathology by inhibiting IL-1β-mediated neuroinflammation
Withaferin a modulation of microglia autophagy mitigates neuroinflammation and enhances cognitive function in POCD
Multimodal and multidomain lesion network mapping enhances prediction of sensorimotor behavior in stroke patients
Aerobic and resistance training enhances endothelial progenitor cell function via upregulation of caveolin-1 in mice with type 2 diabetes
Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen
Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen
Caveolin-1 enhances resveratrol-mediated cytotoxicity and transport in a hepatocellular carcinoma model
Caveolin-2 associates with intracellular chlamydial inclusions independently of caveolin-1
Hyperglycemia enhances brain susceptibility to lipopolysaccharide-induced neuroinflammation via astrocyte reprogramming
Klonierung des muskelspezifischen Caveolin-3 und transkriptionelle Regulation des Caveolin-3-Promoters
Spatial transcriptomics and neurofilament light chain reveal changes in lesion patterns in murine autoimmune neuroinflammation
Complement C3 Enhances LPS-Elicited Neuroinflammation and Neurodegeneration Via the Mac1/NOX2 Pathway
NLRP3 ablation enhances tolerance in heat stroke pathology by inhibiting IL-1β-mediated neuroinflammation
Withaferin a modulation of microglia autophagy mitigates neuroinflammation and enhances cognitive function in POCD
Multimodal and multidomain lesion network mapping enhances prediction of sensorimotor behavior in stroke patients
Aerobic and resistance training enhances endothelial progenitor cell function via upregulation of caveolin-1 in mice with type 2 diabetes
Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen