Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1742-2094
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice ; volume:11 ; number:1 ; day:3 ; month:3 ; year:2014 ; pages:1-14 ; date:12.2014
Journal of neuroinflammation ; 11, Heft 1 (3.3.2014), 1-14, 12.2014
- Urheber
-
Niesman, Ingrid R.
- Beteiligte Personen und Organisationen
-
Schilling, Jan M.
Shapiro, Lee A.
Kellerhals, Sarah E.
Bonds, Jacqueline A.
Kleschevnikov, Alexander M.
Cui, Weihua
Voong, April
Krajewski, Stan
Ali, Sameh S.
Roth, David M.
Patel, Hemal H.
Patel, Piyush M.
Head, Brian P.
SpringerLink (Online service)
- DOI
-
10.1186/1742-2094-11-39
- URN
-
urn:nbn:de:1111-2016032337480
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:44 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Niesman, Ingrid R.
- Schilling, Jan M.
- Shapiro, Lee A.
- Kellerhals, Sarah E.
- Bonds, Jacqueline A.
- Kleschevnikov, Alexander M.
- Cui, Weihua
- Voong, April
- Krajewski, Stan
- Ali, Sameh S.
- Roth, David M.
- Patel, Hemal H.
- Patel, Piyush M.
- Head, Brian P.
- SpringerLink (Online service)
Ähnliche Objekte (12)
Caveolin-1 enhances resveratrol-mediated cytotoxicity and transport in a hepatocellular carcinoma model
Caveolin-2 associates with intracellular chlamydial inclusions independently of caveolin-1
Hyperglycemia enhances brain susceptibility to lipopolysaccharide-induced neuroinflammation via astrocyte reprogramming
Klonierung des muskelspezifischen Caveolin-3 und transkriptionelle Regulation des Caveolin-3-Promoters
Complement C3 Enhances LPS-Elicited Neuroinflammation and Neurodegeneration Via the Mac1/NOX2 Pathway
Spatial transcriptomics and neurofilament light chain reveal changes in lesion patterns in murine autoimmune neuroinflammation
Withaferin a modulation of microglia autophagy mitigates neuroinflammation and enhances cognitive function in POCD
NLRP3 ablation enhances tolerance in heat stroke pathology by inhibiting IL-1β-mediated neuroinflammation
Multimodal and multidomain lesion network mapping enhances prediction of sensorimotor behavior in stroke patients
Aerobic and resistance training enhances endothelial progenitor cell function via upregulation of caveolin-1 in mice with type 2 diabetes
Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen
Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen
Caveolin-1 enhances resveratrol-mediated cytotoxicity and transport in a hepatocellular carcinoma model
Caveolin-2 associates with intracellular chlamydial inclusions independently of caveolin-1
Hyperglycemia enhances brain susceptibility to lipopolysaccharide-induced neuroinflammation via astrocyte reprogramming
Klonierung des muskelspezifischen Caveolin-3 und transkriptionelle Regulation des Caveolin-3-Promoters
Complement C3 Enhances LPS-Elicited Neuroinflammation and Neurodegeneration Via the Mac1/NOX2 Pathway
Spatial transcriptomics and neurofilament light chain reveal changes in lesion patterns in murine autoimmune neuroinflammation
Withaferin a modulation of microglia autophagy mitigates neuroinflammation and enhances cognitive function in POCD
NLRP3 ablation enhances tolerance in heat stroke pathology by inhibiting IL-1β-mediated neuroinflammation
Multimodal and multidomain lesion network mapping enhances prediction of sensorimotor behavior in stroke patients
Aerobic and resistance training enhances endothelial progenitor cell function via upregulation of caveolin-1 in mice with type 2 diabetes
Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen
Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen
Caveolin-1 enhances resveratrol-mediated cytotoxicity and transport in a hepatocellular carcinoma model
Caveolin-2 associates with intracellular chlamydial inclusions independently of caveolin-1
Hyperglycemia enhances brain susceptibility to lipopolysaccharide-induced neuroinflammation via astrocyte reprogramming
Klonierung des muskelspezifischen Caveolin-3 und transkriptionelle Regulation des Caveolin-3-Promoters
Complement C3 Enhances LPS-Elicited Neuroinflammation and Neurodegeneration Via the Mac1/NOX2 Pathway
Spatial transcriptomics and neurofilament light chain reveal changes in lesion patterns in murine autoimmune neuroinflammation
Withaferin a modulation of microglia autophagy mitigates neuroinflammation and enhances cognitive function in POCD
NLRP3 ablation enhances tolerance in heat stroke pathology by inhibiting IL-1β-mediated neuroinflammation
Multimodal and multidomain lesion network mapping enhances prediction of sensorimotor behavior in stroke patients
Aerobic and resistance training enhances endothelial progenitor cell function via upregulation of caveolin-1 in mice with type 2 diabetes
Mechanismen der Caveolin-Genregulation in neuralen Zellsystemen