- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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The Role of Thioredoxin System in Shank3 Mouse Model of Autism ; volume:74 ; number:4 ; day:30 ; month:9 ; year:2024 ; pages:1-16 ; date:12.2024
Journal of molecular neuroscience ; 74, Heft 4 (30.9.2024), 1-16, 12.2024
- Creator
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Bazbaz, Wisam
Kartawy, Maryam
Hamoudi, Wajeha
Ojha, Shashank Kumar
Khaliulin, Igor
Amal, Haitham
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s12031-024-02270-y
- URN
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urn:nbn:de:101:1-2412152114445.388254848136
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:36 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Bazbaz, Wisam
- Kartawy, Maryam
- Hamoudi, Wajeha
- Ojha, Shashank Kumar
- Khaliulin, Igor
- Amal, Haitham
- SpringerLink (Online service)
Other Objects (12)
Characterization of a novel GFP-SHANK3 transgenic mouse model of autism spectrum disorders
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Altered intestinal morphology and microbiota composition in the autism spectrum disorders associated SHANK3 mouse model
Characterization of the structural and functional impact of autism-associated SHANK3 missense mutations
Association study of SHANK3 gene polymorphisms with autism in Chinese Han population
Shank3 transgenic and prenatal zinc-deficient autism mouse models show convergent and Individual alterations of brain structures in MRI
Unravelling the role of SHANK3 mutations in targeted therapies for autism spectrum disorders
Autism-associated gene shank3 is necessary for social contagion in zebrafish
SHANK3 haploinsufficiency: a “common” but underdiagnosed highly penetrant monogenic cause of autism spectrum disorders
Deletion of the autism-associated protein SHANK3 abolishes structural synaptic plasticity after brain trauma
Disrupted Human–Dog Interbrain Neural Coupling in Autism‐Associated Shank3 Mutant Dogs
Thioredoxin family proteins in the db/db mouse
Characterization of a novel GFP-SHANK3 transgenic mouse model of autism spectrum disorders
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Altered intestinal morphology and microbiota composition in the autism spectrum disorders associated SHANK3 mouse model
Characterization of the structural and functional impact of autism-associated SHANK3 missense mutations
Association study of SHANK3 gene polymorphisms with autism in Chinese Han population
Shank3 transgenic and prenatal zinc-deficient autism mouse models show convergent and Individual alterations of brain structures in MRI
Unravelling the role of SHANK3 mutations in targeted therapies for autism spectrum disorders
Autism-associated gene shank3 is necessary for social contagion in zebrafish
SHANK3 haploinsufficiency: a “common” but underdiagnosed highly penetrant monogenic cause of autism spectrum disorders
Deletion of the autism-associated protein SHANK3 abolishes structural synaptic plasticity after brain trauma
Disrupted Human–Dog Interbrain Neural Coupling in Autism‐Associated Shank3 Mutant Dogs
Thioredoxin family proteins in the db/db mouse
Characterization of a novel GFP-SHANK3 transgenic mouse model of autism spectrum disorders
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Altered intestinal morphology and microbiota composition in the autism spectrum disorders associated SHANK3 mouse model
Characterization of the structural and functional impact of autism-associated SHANK3 missense mutations
Association study of SHANK3 gene polymorphisms with autism in Chinese Han population
Shank3 transgenic and prenatal zinc-deficient autism mouse models show convergent and Individual alterations of brain structures in MRI
Unravelling the role of SHANK3 mutations in targeted therapies for autism spectrum disorders
Autism-associated gene shank3 is necessary for social contagion in zebrafish
SHANK3 haploinsufficiency: a “common” but underdiagnosed highly penetrant monogenic cause of autism spectrum disorders
Deletion of the autism-associated protein SHANK3 abolishes structural synaptic plasticity after brain trauma
Disrupted Human–Dog Interbrain Neural Coupling in Autism‐Associated Shank3 Mutant Dogs