Sestrin-2, a repressor of PDGFRβ signalling, promotes cigarette-smoke-induced pulmonary emphysema in mice and is upregulated in individuals with COPD

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: In: Disease models & mechanisms, 6.2013, Heft 6, S. 1378-1387, doi:10.1242/dmm.013482

Keyword: Atemwegskrankheit
Atemnot

Event: Veröffentlichung

(where): Frankfurt am Main

(who): Universitätsbibliothek Johann Christian Senckenberg

(when): 2013

Creator: Heidler, Juliana
Fysikopoulos, Athanasios
Wempe, Frank
Seimetz, Michael
Bangsow, Thorsten
Tomasovic, Ana
Veit, Florian
Scheibe, Susan
Pichl, Alexandra
Weisel, Friederike
Lloyd, K. C. Kent
Jaksch, Peter
Klepetko, Walter
Weissmann, Norbert
Melchner, Harald von

URN: urn:nbn:de:hebis:30:3-342195

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:02 AM CEST

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Associated

Heidler, Juliana
Fysikopoulos, Athanasios
Wempe, Frank
Seimetz, Michael
Bangsow, Thorsten
Tomasovic, Ana
Veit, Florian
Scheibe, Susan
Pichl, Alexandra
Weisel, Friederike
Lloyd, K. C. Kent
Jaksch, Peter
Klepetko, Walter
Weissmann, Norbert
Melchner, Harald von
Universitätsbibliothek Johann Christian Senckenberg

Time of origin

2013

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Sestrin-2, a repressor of PDGFRβ signalling, promotes cigarette smokeinduced pulmonary emphysema in mice and is upregulated in patients with COPD

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p38 mitogen-activated protein kinase determines the susceptibility to cigarette smoke-induced emphysema in mice

Loss of IL-33 enhances elastase-induced and cigarette smoke extract-induced emphysema in mice

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Sestrin-2, a repressor of PDGFRβ signalling, promotes cigarette-smoke-induced pulmonary emphysema in mice and is upregulated in individuals with COPD

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Other Objects (12)

Comparison between cigarette smoke-induced emphysema and cigarette smoke extract-induced emphysema

Sestrin-2, a repressor of PDGFRβ signalling, promotes cigarette smokeinduced pulmonary emphysema in mice and is upregulated in patients with COPD

Cigarette smoke-induced pulmonary emphysema in scid-mice. Is the acquired immune system required?

p38 mitogen-activated protein kinase determines the susceptibility to cigarette smoke-induced emphysema in mice

Loss of IL-33 enhances elastase-induced and cigarette smoke extract-induced emphysema in mice

Ursolic acid alleviates airway-vessel remodeling and muscle consumption in cigarette smoke-induced emphysema rats

Early-life house dust mite aeroallergen exposure augments cigarette smoke-induced myeloid inflammation and emphysema in mice

CSEO – the Cigarette Smoke Exposure Ontology

RAGE is a Critical Mediator of Pulmonary Oxidative Stress, Alveolar Macrophage Activation and Emphysema in Response to Cigarette Smoke

Emphysema is associated with increased inflammation in lungs of atherosclerosis-prone mice by cigarette smoke: implications in comorbidities of COPD

Transcriptional profiling and targeted proteomics reveals common molecular changes associated with cigarette smoke-induced lung emphysema development in five susceptible mouse strains

Oxidative stress mediates the apoptosis and epigenetic modification of the Bcl-2 promoter via DNMT1 in a cigarette smoke-induced emphysema model

Comparison between cigarette smoke-induced emphysema and cigarette smoke extract-induced emphysema

Sestrin-2, a repressor of PDGFRβ signalling, promotes cigarette smokeinduced pulmonary emphysema in mice and is upregulated in patients with COPD

Cigarette smoke-induced pulmonary emphysema in scid-mice. Is the acquired immune system required?

p38 mitogen-activated protein kinase determines the susceptibility to cigarette smoke-induced emphysema in mice

Loss of IL-33 enhances elastase-induced and cigarette smoke extract-induced emphysema in mice

Ursolic acid alleviates airway-vessel remodeling and muscle consumption in cigarette smoke-induced emphysema rats

Early-life house dust mite aeroallergen exposure augments cigarette smoke-induced myeloid inflammation and emphysema in mice

CSEO – the Cigarette Smoke Exposure Ontology

RAGE is a Critical Mediator of Pulmonary Oxidative Stress, Alveolar Macrophage Activation and Emphysema in Response to Cigarette Smoke

Emphysema is associated with increased inflammation in lungs of atherosclerosis-prone mice by cigarette smoke: implications in comorbidities of COPD

Transcriptional profiling and targeted proteomics reveals common molecular changes associated with cigarette smoke-induced lung emphysema development in five susceptible mouse strains

Oxidative stress mediates the apoptosis and epigenetic modification of the Bcl-2 promoter via DNMT1 in a cigarette smoke-induced emphysema model

Comparison between cigarette smoke-induced emphysema and cigarette smoke extract-induced emphysema

Sestrin-2, a repressor of PDGFRβ signalling, promotes cigarette smokeinduced pulmonary emphysema in mice and is upregulated in patients with COPD

Cigarette smoke-induced pulmonary emphysema in scid-mice. Is the acquired immune system required?

p38 mitogen-activated protein kinase determines the susceptibility to cigarette smoke-induced emphysema in mice

Loss of IL-33 enhances elastase-induced and cigarette smoke extract-induced emphysema in mice

Ursolic acid alleviates airway-vessel remodeling and muscle consumption in cigarette smoke-induced emphysema rats

Early-life house dust mite aeroallergen exposure augments cigarette smoke-induced myeloid inflammation and emphysema in mice

CSEO – the Cigarette Smoke Exposure Ontology

RAGE is a Critical Mediator of Pulmonary Oxidative Stress, Alveolar Macrophage Activation and Emphysema in Response to Cigarette Smoke

Emphysema is associated with increased inflammation in lungs of atherosclerosis-prone mice by cigarette smoke: implications in comorbidities of COPD

Transcriptional profiling and targeted proteomics reveals common molecular changes associated with cigarette smoke-induced lung emphysema development in five susceptible mouse strains

Oxidative stress mediates the apoptosis and epigenetic modification of the Bcl-2 promoter via DNMT1 in a cigarette smoke-induced emphysema model

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