Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies ; volume:9 ; number:7 ; year:2017 ; pages:853-855 ; extent:3
EMBO molecular medicine / European Molecular Biology Organization ; 9, Heft 7 (2017), 853-855 (gesamt 3)

Creator: Balendra, Rubika
Moens, Thomas G.
Isaacs, Adrian M.

DOI: 10.15252/emmm.201707848

URN: urn:nbn:de:101:1-2022092905324360154212

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:23 AM CEST

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Balendra, Rubika
Moens, Thomas G.
Isaacs, Adrian M.

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Specific biomarkers for C9orf72 FTD/ALS could expedite the journey towards effective therapies

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Other Objects (12)

A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma

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A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD

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The ALS/FTD-related C9orf72 hexanucleotide repeat expansion forms RNA condensates through multimolecular G-quadruplexes

CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts

Correction to: Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD

A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma

Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit

A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD

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Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD

Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD

Phenylalanine-tRNA aminoacylation is compromised by ALS/FTD-associated C9orf72 C4G2 repeat RNA

The ALS/FTD-related C9orf72 hexanucleotide repeat expansion forms RNA condensates through multimolecular G-quadruplexes

CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts

Correction to: Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD

A familial FTD associated with C9orf72 repeat expansion and dysplastic gangliocytoma

Roadmap for C9ORF72 in Frontotemporal Dementia and Amyotrophic Lateral Sclerosis: Report on the C9ORF72 FTD/ALS Summit

A C9ORF72 BAC mouse model recapitulates key epigenetic perturbations of ALS/FTD

Nuclear lamina invaginations are not a pathological feature of C9orf72 ALS/FTD

Aktive und passive anti-GA Immuntherapie für C9orf72 ALS/FTD im Mausmodell

RNA dependent suppression of C9orf72 ALS/FTD associated neurodegeneration by Matrin-3

Targeting RNA G‐quadruplexes as new treatment strategy for C9orf72 ALS/FTD

Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD

Phenylalanine-tRNA aminoacylation is compromised by ALS/FTD-associated C9orf72 C4G2 repeat RNA

The ALS/FTD-related C9orf72 hexanucleotide repeat expansion forms RNA condensates through multimolecular G-quadruplexes

CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts

Correction to: Divergence, Convergence, and Therapeutic Implications: A Cell Biology Perspective of C9ORF72-ALS/FTD

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