Activation of the hypoxia response protects mice from amyloid-β accumulation
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1420-9071
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Activation of the hypoxia response protects mice from amyloid-β accumulation ; volume:79 ; number:8 ; day:19 ; month:7 ; year:2022 ; pages:1-15 ; date:8.2022
Cellular and molecular life sciences ; 79, Heft 8 (19.7.2022), 1-15, 8.2022
- Urheber
-
Ollonen, Teemu
Kurkela, Margareta
Laitakari, Anna
Sakko, Samuli
Koivisto, Henna
Myllyharju, Johanna
Tanila, Heikki
Serpi, Raisa
Koivunen, Peppi
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1007/s00018-022-04460-6
- URN
-
urn:nbn:de:101:1-2022111021063503561213
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:23 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Ollonen, Teemu
- Kurkela, Margareta
- Laitakari, Anna
- Sakko, Samuli
- Koivisto, Henna
- Myllyharju, Johanna
- Tanila, Heikki
- Serpi, Raisa
- Koivunen, Peppi
- SpringerLink (Online service)
Ähnliche Objekte (12)
Crocetin attenuates inflammation and amyloid-β accumulation in APPsw transgenic mice
Origins of amyloid-β
Selective amyloid-β lowering agents
Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
![Development of conformation specific Amyloid-β [Amyloid-beta] antibodies](/assets/placeholder/searchResultMediaNoDigitisedMedia.png)
Development of conformation specific Amyloid-β [Amyloid-beta] antibodies
Senescence-related impairment of autophagy induces toxic intraneuronal amyloid-β accumulation in a mouse model of amyloid pathology
Memory Impairment in Estrogen Receptor α Knockout Mice Through Accumulation of Amyloid-β Peptides
Amyloid-β accumulation in the CNS in human growth hormone recipients in the UK
Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery
The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro
Copper mediated amyloid-β binding to Transthyretin
Accumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neurons
Crocetin attenuates inflammation and amyloid-β accumulation in APPsw transgenic mice
Origins of amyloid-β
Selective amyloid-β lowering agents
Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
Development of conformation specific Amyloid-β [Amyloid-beta] antibodies
Senescence-related impairment of autophagy induces toxic intraneuronal amyloid-β accumulation in a mouse model of amyloid pathology
Memory Impairment in Estrogen Receptor α Knockout Mice Through Accumulation of Amyloid-β Peptides
Amyloid-β accumulation in the CNS in human growth hormone recipients in the UK
Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery
The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro
Copper mediated amyloid-β binding to Transthyretin
Accumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neurons
Crocetin attenuates inflammation and amyloid-β accumulation in APPsw transgenic mice
Origins of amyloid-β
Selective amyloid-β lowering agents
Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism
Development of conformation specific Amyloid-β [Amyloid-beta] antibodies
Senescence-related impairment of autophagy induces toxic intraneuronal amyloid-β accumulation in a mouse model of amyloid pathology
Memory Impairment in Estrogen Receptor α Knockout Mice Through Accumulation of Amyloid-β Peptides
Amyloid-β accumulation in the CNS in human growth hormone recipients in the UK
Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery
The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro
Copper mediated amyloid-β binding to Transthyretin