Activation of the hypoxia response protects mice from amyloid-β accumulation

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1420-9071

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Activation of the hypoxia response protects mice from amyloid-β accumulation ; volume:79 ; number:8 ; day:19 ; month:7 ; year:2022 ; pages:1-15 ; date:8.2022
Cellular and molecular life sciences ; 79, Heft 8 (19.7.2022), 1-15, 8.2022

Creator: Ollonen, Teemu
Kurkela, Margareta
Laitakari, Anna
Sakko, Samuli
Koivisto, Henna
Myllyharju, Johanna
Tanila, Heikki
Serpi, Raisa
Koivunen, Peppi

Contributor: SpringerLink (Online service)

DOI: 10.1007/s00018-022-04460-6

URN: urn:nbn:de:101:1-2022111021063503561213

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:23 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Ollonen, Teemu
Kurkela, Margareta
Laitakari, Anna
Sakko, Samuli
Koivisto, Henna
Myllyharju, Johanna
Tanila, Heikki
Serpi, Raisa
Koivunen, Peppi
SpringerLink (Online service)

Other Objects (12)

Crocetin attenuates inflammation and amyloid-β accumulation in APPsw transgenic mice

Origins of amyloid-β

Selective amyloid-β lowering agents

Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

Hochschulschrift

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

Senescence-related impairment of autophagy induces toxic intraneuronal amyloid-β accumulation in a mouse model of amyloid pathology

Amyloid-β accumulation in the CNS in human growth hormone recipients in the UK

Memory Impairment in Estrogen Receptor α Knockout Mice Through Accumulation of Amyloid-β Peptides

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Accumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neurons

Crocetin attenuates inflammation and amyloid-β accumulation in APPsw transgenic mice

Origins of amyloid-β

Selective amyloid-β lowering agents

Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

Hochschulschrift

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

Senescence-related impairment of autophagy induces toxic intraneuronal amyloid-β accumulation in a mouse model of amyloid pathology

Amyloid-β accumulation in the CNS in human growth hormone recipients in the UK

Memory Impairment in Estrogen Receptor α Knockout Mice Through Accumulation of Amyloid-β Peptides

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Accumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neurons

Crocetin attenuates inflammation and amyloid-β accumulation in APPsw transgenic mice

Origins of amyloid-β

Selective amyloid-β lowering agents

Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

Hochschulschrift

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

Senescence-related impairment of autophagy induces toxic intraneuronal amyloid-β accumulation in a mouse model of amyloid pathology

Amyloid-β accumulation in the CNS in human growth hormone recipients in the UK

Memory Impairment in Estrogen Receptor α Knockout Mice Through Accumulation of Amyloid-β Peptides

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Accumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neurons

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

Activation of the hypoxia response protects mice from amyloid-β accumulation

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Crocetin attenuates inflammation and amyloid-β accumulation in APPsw transgenic mice

Origins of amyloid-β

Selective amyloid-β lowering agents

Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

Senescence-related impairment of autophagy induces toxic intraneuronal amyloid-β accumulation in a mouse model of amyloid pathology

Amyloid-β accumulation in the CNS in human growth hormone recipients in the UK

Memory Impairment in Estrogen Receptor α Knockout Mice Through Accumulation of Amyloid-β Peptides

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Accumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neurons

Crocetin attenuates inflammation and amyloid-β accumulation in APPsw transgenic mice

Origins of amyloid-β

Selective amyloid-β lowering agents

Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

Senescence-related impairment of autophagy induces toxic intraneuronal amyloid-β accumulation in a mouse model of amyloid pathology

Amyloid-β accumulation in the CNS in human growth hormone recipients in the UK

Memory Impairment in Estrogen Receptor α Knockout Mice Through Accumulation of Amyloid-β Peptides

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Accumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neurons

Crocetin attenuates inflammation and amyloid-β accumulation in APPsw transgenic mice

Origins of amyloid-β

Selective amyloid-β lowering agents

Amyloid-β accumulation in human astrocytes induces mitochondrial disruption and changed energy metabolism

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

Senescence-related impairment of autophagy induces toxic intraneuronal amyloid-β accumulation in a mouse model of amyloid pathology

Amyloid-β accumulation in the CNS in human growth hormone recipients in the UK

Memory Impairment in Estrogen Receptor α Knockout Mice Through Accumulation of Amyloid-β Peptides

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

The modulating effect of soluble amyloid-β dimers on amyloid-β aggregation in vivo and in vitro

Copper mediated amyloid-β binding to Transthyretin

Accumulation of amyloid-β by astrocytes result in enlarged endosomes and microvesicle-induced apoptosis of neurons

Related objects

Reset password