PDGF regulated migration of mesenchymal stem cells towards malignancy acts via the PI3K signaling pathway
- Weitere Titel
-
PDGF regulierte Migration von mesenchymalen Stammzellen in Richtung Tumorzellen über den PI3K Signalweg
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Deutsch
- Anmerkungen
-
Regensburg, Universität Regensburg, Dissertation, 2019
- Schlagwort
-
Mesenchymal stem cells
- Ereignis
-
Veröffentlichung
- (wo)
-
Regensburg
- (wer)
-
Universitätsbibliothek Regensburg
- (wann)
-
2019
- Urheber
- Beteiligte Personen und Organisationen
-
Prantl, Lukas
- URN
-
urn:nbn:de:bvb:355-epub-410468
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
25.03.2025, 13:52 MEZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Salha, Sonia
- Prantl, Lukas
- Universitätsbibliothek Regensburg
Entstanden
- 2019
Ähnliche Objekte (12)
Co-regulated malignancy associated genes in multiple cancer progression models
Chemotherapy and urological malignancy
Combination therapy in urological malignancy
Autocrine and paracrine effects of mechanically regulated matrix metalloproteases in human mesenchymal stem cells
Osteoclasts control osteoblast chemotaxis via PDGF-BB/PDGF receptor beta signaling
Biological responses to PDGF-AA versus PDGF-CC in renal fibroblasts
Hypercalcemia of malignancy : with 17 tables
Immunhistochemischer Nachweis von PDGF B, PDGF-β-Rezeptoren [PDGF-beta-Rezeptoren] und IGF-I-Rezeptoren während der Atherogenese beim Menschen
PI3K-Inhibition im Pankreaskarzinommodell
Exclusive inhibition of PI3K/Akt/mTOR signaling is not sufficient to prevent PDGF-mediated effects on glycolysis and proliferation in colorectal cancer
Inhibiting PI3K–AKT–mTOR Signaling in Multiple Myeloma-Associated Mesenchymal Stem Cells Impedes the Proliferation of Multiple Myeloma Cells
Leukocyte-reduced platelet-rich plasma stimulates the in vitro proliferation of adipose-tissue derived mesenchymal stem cells depending on PDGF signaling.
Co-regulated malignancy associated genes in multiple cancer progression models
Chemotherapy and urological malignancy
Combination therapy in urological malignancy
Autocrine and paracrine effects of mechanically regulated matrix metalloproteases in human mesenchymal stem cells
Osteoclasts control osteoblast chemotaxis via PDGF-BB/PDGF receptor beta signaling
Biological responses to PDGF-AA versus PDGF-CC in renal fibroblasts
Hypercalcemia of malignancy : with 17 tables
Immunhistochemischer Nachweis von PDGF B, PDGF-β-Rezeptoren [PDGF-beta-Rezeptoren] und IGF-I-Rezeptoren während der Atherogenese beim Menschen
PI3K-Inhibition im Pankreaskarzinommodell
Exclusive inhibition of PI3K/Akt/mTOR signaling is not sufficient to prevent PDGF-mediated effects on glycolysis and proliferation in colorectal cancer
Inhibiting PI3K–AKT–mTOR Signaling in Multiple Myeloma-Associated Mesenchymal Stem Cells Impedes the Proliferation of Multiple Myeloma Cells
Leukocyte-reduced platelet-rich plasma stimulates the in vitro proliferation of adipose-tissue derived mesenchymal stem cells depending on PDGF signaling.
Co-regulated malignancy associated genes in multiple cancer progression models
Chemotherapy and urological malignancy
Combination therapy in urological malignancy
Autocrine and paracrine effects of mechanically regulated matrix metalloproteases in human mesenchymal stem cells
Osteoclasts control osteoblast chemotaxis via PDGF-BB/PDGF receptor beta signaling
Biological responses to PDGF-AA versus PDGF-CC in renal fibroblasts
Hypercalcemia of malignancy : with 17 tables
Immunhistochemischer Nachweis von PDGF B, PDGF-β-Rezeptoren [PDGF-beta-Rezeptoren] und IGF-I-Rezeptoren während der Atherogenese beim Menschen
PI3K-Inhibition im Pankreaskarzinommodell
Exclusive inhibition of PI3K/Akt/mTOR signaling is not sufficient to prevent PDGF-mediated effects on glycolysis and proliferation in colorectal cancer
Inhibiting PI3K–AKT–mTOR Signaling in Multiple Myeloma-Associated Mesenchymal Stem Cells Impedes the Proliferation of Multiple Myeloma Cells