Hochschulschrift
TGF-β,Smad [TGF-beta,Smad] signaling in hepatic stellate cells and during liver fibrogenesis
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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Aachen, Techn. Hochsch., Diss., 2005
- Keyword
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Leber
Gallenwege
Zellzyklus
Signaltransduktion
- Creator
- URN
-
urn:nbn:de:hbz:82-opus-16101
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:50 PM CET
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Object type
- Hochschulschrift
Associated
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TGF-β,Smad [TGF-beta,Smad] signaling in hepatic stellate cells and during liver fibrogenesis
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Effects of nerve growth factor on TGF-β-Smad [TGF-beta-Smad] signal transduction in PC12 cells
Interaktion zwischen TGF-β-Smad [TGF-beta-Smad] 3- und dem Glukokortikoidrezeptor vermittelter Signalweg in hepatischen Sternzellen
Systematic analysis of TGF-β-Smad [TGF-beta-Smad] signaling and (loss of) cytostatic outcome in human hepatocellular carcinoma cell lines
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Overexpression of Endoglin Modulates TGF-β1-Signalling Pathways in a Novel Immortalized Mouse Hepatic Stellate Cell Line
Hepatitis C-virus replicating hepatocytes induce profibrogenic gene expression in hepatic stellate cells via release of TGF-beta
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Induction of smad-dependent and -independent pathways by TGF-βs [TGF-beta-s] in human endometrial and endometriotic cells
Effect of progesterone on Smad signaling and TGF-β/Smad-regulated genes in lung epithelial cells
TGF-β,Smad [TGF-beta,Smad] signaling in hepatic stellate cells and during liver fibrogenesis
Effects of nerve growth factor on TGF-β-Smad [TGF-beta-Smad] signal transduction in PC12 cells
Effects of nerve growth factor on TGF-β-Smad [TGF-beta-Smad] signal transduction in PC12 cells
Interaktion zwischen TGF-β-Smad [TGF-beta-Smad] 3- und dem Glukokortikoidrezeptor vermittelter Signalweg in hepatischen Sternzellen
Systematic analysis of TGF-β-Smad [TGF-beta-Smad] signaling and (loss of) cytostatic outcome in human hepatocellular carcinoma cell lines
Einfluss von Prolyl-Hydroxylase-Inhibitoren auf den TGF-β/Smad-Signalweg [TGF-Beta/Smad-Signalweg] und die Migration humaner primärer Tubulusepithelzellen
Overexpression of Endoglin Modulates TGF-β1-Signalling Pathways in a Novel Immortalized Mouse Hepatic Stellate Cell Line
Hepatitis C-virus replicating hepatocytes induce profibrogenic gene expression in hepatic stellate cells via release of TGF-beta
Regulation of T-cell differentiation by the TGF-β [TGF-beta] inhibitor Smad7 and non-Smad pathways
Fibroproliferative Eigenschaften von Fibroblasten, abhängig von dem TGF-β-Signaltransduktionsweg [TGF-beta-Signaltransduktionsweg] und blockiert durch Smad 7
Induction of smad-dependent and -independent pathways by TGF-βs [TGF-beta-s] in human endometrial and endometriotic cells
Effect of progesterone on Smad signaling and TGF-β/Smad-regulated genes in lung epithelial cells
TGF-β,Smad [TGF-beta,Smad] signaling in hepatic stellate cells and during liver fibrogenesis
Effects of nerve growth factor on TGF-β-Smad [TGF-beta-Smad] signal transduction in PC12 cells
Effects of nerve growth factor on TGF-β-Smad [TGF-beta-Smad] signal transduction in PC12 cells
Interaktion zwischen TGF-β-Smad [TGF-beta-Smad] 3- und dem Glukokortikoidrezeptor vermittelter Signalweg in hepatischen Sternzellen
Systematic analysis of TGF-β-Smad [TGF-beta-Smad] signaling and (loss of) cytostatic outcome in human hepatocellular carcinoma cell lines
Einfluss von Prolyl-Hydroxylase-Inhibitoren auf den TGF-β/Smad-Signalweg [TGF-Beta/Smad-Signalweg] und die Migration humaner primärer Tubulusepithelzellen
Overexpression of Endoglin Modulates TGF-β1-Signalling Pathways in a Novel Immortalized Mouse Hepatic Stellate Cell Line
Hepatitis C-virus replicating hepatocytes induce profibrogenic gene expression in hepatic stellate cells via release of TGF-beta
Regulation of T-cell differentiation by the TGF-β [TGF-beta] inhibitor Smad7 and non-Smad pathways
Fibroproliferative Eigenschaften von Fibroblasten, abhängig von dem TGF-β-Signaltransduktionsweg [TGF-beta-Signaltransduktionsweg] und blockiert durch Smad 7
Induction of smad-dependent and -independent pathways by TGF-βs [TGF-beta-s] in human endometrial and endometriotic cells