TDP-43 regulates LC3ylation in neural tissue through ATG4B cryptic splicing inhibition

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: TDP-43 regulates LC3ylation in neural tissue through ATG4B cryptic splicing inhibition ; volume:148 ; number:1 ; day:21 ; month:9 ; year:2024 ; pages:1-22 ; date:12.2024
Acta neuropathologica ; 148, Heft 1 (21.9.2024), 1-22, 12.2024

Classification: Medizin, Gesundheit

Creator: Torres, Pascual
Rico-Rios, Santiago
Ceron-Codorniu, Miriam
Santacreu-Vilaseca, Marta
Seoane-Miraz, David
Jad, Yahya
Ayala, Victòria
Mariño, Guillermo
Beltran, Maria
Miralles, Maria P.
Andrés-Benito, Pol
Fernandez-Irigoyen, Joaquin
Santamaria, Enrique
López-Otín, Carlos
Soler, Rosa M.
Povedano, Monica
Ferrer, Isidro
Pamplona, Reinald
Wood, Matthew J. A.
Varela, Miguel A.
Portero-Otin, Manuel

Contributor: SpringerLink (Online service)

DOI: 10.1007/s00401-024-02780-4

URN: urn:nbn:de:101:1-2412050706050.830810377716

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:34 AM CEST

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Associated

Torres, Pascual
Rico-Rios, Santiago
Ceron-Codorniu, Miriam
Santacreu-Vilaseca, Marta
Seoane-Miraz, David
Jad, Yahya
Ayala, Victòria
Mariño, Guillermo
Beltran, Maria
Miralles, Maria P.
Andrés-Benito, Pol
Fernandez-Irigoyen, Joaquin
Santamaria, Enrique
López-Otín, Carlos
Soler, Rosa M.
Povedano, Monica
Ferrer, Isidro
Pamplona, Reinald
Wood, Matthew J. A.
Varela, Miguel A.
Portero-Otin, Manuel
SpringerLink (Online service)

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Quantitative analysis of cryptic splicing associated with TDP-43 depletion

SUMOylation Regulates TDP-43 Splicing Activity and Nucleocytoplasmic Distribution

Tdp-43 cryptic exons are highly variable between cell types

Cryptic splicing of stathmin-2 and UNC13A mRNAs is a pathological hallmark of TDP-43-associated Alzheimer’s disease

TDP-43 regulates cancer-associated microRNAs

HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

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The N-terminal dimerization is required for TDP-43 splicing activity

RNA aptamer reveals nuclear TDP-43 pathology is an early aggregation event that coincides with STMN-2 cryptic splicing and precedes clinical manifestation in ALS

Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathy

Impaired human hematopoiesis due to a cryptic intronic GATA1 splicing mutation

Quantitative analysis of cryptic splicing associated with TDP-43 depletion

SUMOylation Regulates TDP-43 Splicing Activity and Nucleocytoplasmic Distribution

Tdp-43 cryptic exons are highly variable between cell types

Cryptic splicing of stathmin-2 and UNC13A mRNAs is a pathological hallmark of TDP-43-associated Alzheimer’s disease

TDP-43 regulates cancer-associated microRNAs

HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

Correction: HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

The N-terminal dimerization is required for TDP-43 splicing activity

RNA aptamer reveals nuclear TDP-43 pathology is an early aggregation event that coincides with STMN-2 cryptic splicing and precedes clinical manifestation in ALS

Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathy

Impaired human hematopoiesis due to a cryptic intronic GATA1 splicing mutation

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TDP-43 regulates LC3ylation in neural tissue through ATG4B cryptic splicing inhibition

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Other Objects (12)

Quantitative analysis of cryptic splicing associated with TDP-43 depletion

SUMOylation Regulates TDP-43 Splicing Activity and Nucleocytoplasmic Distribution

Tdp-43 cryptic exons are highly variable between cell types

Cryptic splicing of stathmin-2 and UNC13A mRNAs is a pathological hallmark of TDP-43-associated Alzheimer’s disease

TDP-43 regulates cancer-associated microRNAs

HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

Correction: HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

The N-terminal dimerization is required for TDP-43 splicing activity

RNA aptamer reveals nuclear TDP-43 pathology is an early aggregation event that coincides with STMN-2 cryptic splicing and precedes clinical manifestation in ALS

Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathy

Impaired human hematopoiesis due to a cryptic intronic GATA1 splicing mutation

Quantitative analysis of cryptic splicing associated with TDP-43 depletion

SUMOylation Regulates TDP-43 Splicing Activity and Nucleocytoplasmic Distribution

Tdp-43 cryptic exons are highly variable between cell types

Cryptic splicing of stathmin-2 and UNC13A mRNAs is a pathological hallmark of TDP-43-associated Alzheimer’s disease

TDP-43 regulates cancer-associated microRNAs

HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

Correction: HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

The N-terminal dimerization is required for TDP-43 splicing activity

RNA aptamer reveals nuclear TDP-43 pathology is an early aggregation event that coincides with STMN-2 cryptic splicing and precedes clinical manifestation in ALS

Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathy

Impaired human hematopoiesis due to a cryptic intronic GATA1 splicing mutation

Quantitative analysis of cryptic splicing associated with TDP-43 depletion

SUMOylation Regulates TDP-43 Splicing Activity and Nucleocytoplasmic Distribution

Tdp-43 cryptic exons are highly variable between cell types

Cryptic splicing of stathmin-2 and UNC13A mRNAs is a pathological hallmark of TDP-43-associated Alzheimer’s disease

TDP-43 regulates cancer-associated microRNAs

HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

TDP-43-regulated cryptic RNAs accumulate in Alzheimer’s disease brains

Correction: HDGFL2 cryptic proteins report presence of TDP-43 pathology in neurodegenerative diseases

The N-terminal dimerization is required for TDP-43 splicing activity

RNA aptamer reveals nuclear TDP-43 pathology is an early aggregation event that coincides with STMN-2 cryptic splicing and precedes clinical manifestation in ALS

Alternatively spliced ELAVL3 cryptic exon 4a causes ELAVL3 downregulation in ALS TDP-43 proteinopathy

Impaired human hematopoiesis due to a cryptic intronic GATA1 splicing mutation

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