- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1471-2199
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Rad9 modulates the P21 WAF1 pathway by direct association with p53 ; volume:8 ; number:1 ; day:21 ; month:5 ; year:2007 ; pages:1-10 ; date:12.2007
BMC molecular biology ; 8, Heft 1 (21.5.2007), 1-10, 12.2007
- Classification
-
Biowissenschaften, Biologie
- Creator
-
Ishikawa, Kazuhiro
Ishii, Hideshi
Murakumo, Yoshiki
Mimori, Koshi
Kobayashi, Masahiko
Yamamoto, Ken-ichi
Mori, Masaki
Nishino, Hiroshi
Furukawa, Yusuke
Ichimura, Keiichi
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/1471-2199-8-37
- URN
-
urn:nbn:de:101:1-2019073019222000119824
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:29 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ishikawa, Kazuhiro
- Ishii, Hideshi
- Murakumo, Yoshiki
- Mimori, Koshi
- Kobayashi, Masahiko
- Yamamoto, Ken-ichi
- Mori, Masaki
- Nishino, Hiroshi
- Furukawa, Yusuke
- Ichimura, Keiichi
- SpringerLink (Online service)
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ACSL1 promotes imatinib-induced chronic myeloid leukemia cell senescence by regulating SIRT1/p53/p21 pathway
Synergistic anticancer effect of combined crocetin and cisplatin on KYSE-150 cells via p53/p21 pathway
JNK inhibitor IX restrains pancreatic cancer through p53 and p21
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SPRR2A enhances p53 deacetylation through HDAC1 and down regulates p21 promoter activity
Mutationen des Tumorsuppressorgens p53 beim Magenkarzinom : ein Vergleich zur p53 und p21 Expression sowie zu klinisch-pathologischen Parametern
UBE2C promotes LUAD progression by ubiquitin-dependent degradation of p53 to inactivate the p53/p21 signaling pathway
PIK3R3 inhibits cell senescence through p53/p21 signaling
SARS-COV-2 spike protein promotes RPE cell senescence via the ROS/P53/P21 pathway
ACSL1 promotes imatinib-induced chronic myeloid leukemia cell senescence by regulating SIRT1/p53/p21 pathway
Synergistic anticancer effect of combined crocetin and cisplatin on KYSE-150 cells via p53/p21 pathway
JNK inhibitor IX restrains pancreatic cancer through p53 and p21
ETV4 promotes late development of prostatic intraepithelial neoplasia and cell proliferation through direct and p53-mediated downregulation of p21
A RUNX-targeted gene switch-off approach modulates the BIRC5/PIF1-p21 pathway and reduces glioblastoma growth in mice
MAGE-A-, p53- und p21-Expression als prognostischer Marker bei oralen Plattenepithelkarzinomen
Die Tumorsuppressorgenprodukte p53, p21 und p16 beim Mammakarzinom : Korrelationen mit klassischen Prognosefaktoren
SPRR2A enhances p53 deacetylation through HDAC1 and down regulates p21 promoter activity
Mutationen des Tumorsuppressorgens p53 beim Magenkarzinom : ein Vergleich zur p53 und p21 Expression sowie zu klinisch-pathologischen Parametern
UBE2C promotes LUAD progression by ubiquitin-dependent degradation of p53 to inactivate the p53/p21 signaling pathway
PIK3R3 inhibits cell senescence through p53/p21 signaling
SARS-COV-2 spike protein promotes RPE cell senescence via the ROS/P53/P21 pathway
ACSL1 promotes imatinib-induced chronic myeloid leukemia cell senescence by regulating SIRT1/p53/p21 pathway
Synergistic anticancer effect of combined crocetin and cisplatin on KYSE-150 cells via p53/p21 pathway
JNK inhibitor IX restrains pancreatic cancer through p53 and p21
ETV4 promotes late development of prostatic intraepithelial neoplasia and cell proliferation through direct and p53-mediated downregulation of p21
A RUNX-targeted gene switch-off approach modulates the BIRC5/PIF1-p21 pathway and reduces glioblastoma growth in mice
MAGE-A-, p53- und p21-Expression als prognostischer Marker bei oralen Plattenepithelkarzinomen
Die Tumorsuppressorgenprodukte p53, p21 und p16 beim Mammakarzinom : Korrelationen mit klassischen Prognosefaktoren
SPRR2A enhances p53 deacetylation through HDAC1 and down regulates p21 promoter activity